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GeneRIF024108.RAC7 ibMhMSylYBKCzYVJ7Y7JMbZiaNJ5F5Dpu19WuRW4
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

GeneRIF024192.RABhNoGf9AGHz5Gt4P0ehecOgXCGKZAxWuyrVllsJlVVY
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

GeneRIF024302.RAB-3UbtF88mkAdtQ8QPIk3YUksfqLkMb9QvBt3qTHttE
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

GeneRIF024705.RACD-bVF2RQQTglCinq504mhcGF--Mw8iexZGgjAo9L80
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

GeneRIF024790.RAD-liz248N0PlkLbTh9cFvkICnE8Xl1aoZkV04H5ES M
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

GeneRIF024931.RABCiMyv0DrEfpikE9dmnM 8j1PvioBaXfuCYen2Vyzy8
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

GeneRIF025095.RABTe3qq6Bxh-4Wge 1TXp3x7Gz9cqbxsybq2fH2mKyBk
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

GeneRIF025123.RAAc1482P7ZorkVXxc2ZRt4mVmIwO9G7xKseHQQ3tEpuA
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

GeneRIF025242.RACjYP3mvdduAMoKsTngfBO7uZ7H6jZfAFTfgNjzrQ98c
has assertion: assertion
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-06-16T13:47:00Z
is part of: NanopubsFromGeneRIF
created by: http://orcid.org/0000-0002-1267-0234
version: 1.3

RAPzQ9qAmFQA8RB1pTuflZdpbkSsnHsunb s691dxfB-8
has assertion: p(MGI:Vav1) -> kin(p(MGI:Akt1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.010+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(MGI:Vav1) -> kin(p(MGI:Akt1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11340169

BEL Framework Large Corpus Document
description: Approximately 61,000 statements.
rights: Copyright (c) 2011-2012, Selventa. All rights reserved.
authored by: Selventa
version: 1.4

5
value: Modified assertion
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11340169

RAO7aIrzK3wB4joqto4mdwQdqdTZybLFo2sEbykGRkR7s
has assertion: cat(p(MGI:Vav1)) -> p(MGI:Plcg1,pmod(P,Y))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.013+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

cat(p(MGI:Vav1)) -> p(MGI:Plcg1,pmod(P,Y))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11340169

5
value: Modified assertion
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11340169

RAP5z6dSEoc2yv6-wy sLfr-KN8r9 caYZocytR8-QtU
has assertion: a(CHEBI:estradiol) -> p(MGI:Raf1,pmod(P,S,259))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.029+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:estradiol) -> p(MGI:Raf1,pmod(P,S,259))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11341776

3
value: We demonstrated that lung myofibroblasts, incubated in the presence of E2, showed a rapid phosphorylation on serine-259 of Raf1 and tyrosine-204 of ERK1/2 MAP kinase at 15 min, by approximately 3- and 5-fold, respectively
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11341776

RAMDq Ur5BcKOe6b85eUq63zafaiP3tu1O5murX8ICI1I
has assertion: kin(p(HGNC:MAPK3)) => p(HGNC:HSPB8,pmod(P,S,27))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.034+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(HGNC:MAPK3)) => p(HGNC:HSPB8,pmod(P,S,27))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11342557

5
value: PhosphoElm data from PMID 15212693
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11342557

RAKj59OvXakOykOlVwITQNy7q75slzBk-eHmU31VdCVIY
has assertion: kin(p(PFH:"PRKC Family")) => p(HGNC:HSPB8,pmod(P,S,14))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.037+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(PFH:"PRKC Family")) => p(HGNC:HSPB8,pmod(P,S,14))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11342557

5
value: PhosphoElm data from PMID 15212693
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11342557

RAKqyuiVgJB4jwX3HTLEo6FqnUrFVQVbqpuCrtFjv5MgY
has assertion: kin(p(MGI:Pik3ca)) -> kin(p(MGI:Akt1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.040+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(MGI:Pik3ca)) -> kin(p(MGI:Akt1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11342583

6
value: Modified assertion
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11342583

RAPXMlNYi88QbHZdJqoUKbxrc5-dgs3pkPHJOjcw dMzg
has assertion: bp(GO:"response to hypoxia") -> r(HGNC:TFF3)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.042+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

bp(GO:"response to hypoxia") -> r(HGNC:TFF3)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11342587

3
value: Hypoxia resulted in parallel induction of ITF mRNA and protein.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11342587

RAMKiJxOQ7uve19Qgf3PmreOYFsom-ov48X6wbV878uXw
has assertion: a(SCHEM:Dinoprostone) -> r(MGI:S100a8)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.048+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(SCHEM:Dinoprostone) -> r(MGI:S100a8)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11342660

3
value: IL-10 decreased the optimal time of mRNA expression induced by LPS from 24 to 8 h. Blocking experiments indicated that endogenous IL-10 contributes to gene induction by LPS. Cooperation between IL-10 and LPS was not due to altered mRNA stability but was dependent on de novo protein synthesis. The region of the promoter involved was localized to a 178-bp fragment flanking the transcription start site of the gene. This region was also responsible for the suppressive effects of IL-4 and IL-13. Forskolin, CTP-cAMP, and PGE(2) also enhanced LPS- and IFN-induced A8 mRNA,
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11342660

RAKejiOVqyjI3lsvsDcAI53fzrpNUMBfqvB6Olv2-S2R8
has assertion: a(SCHEM:"cyclic AMP") -> r(MGI:S100a8)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.050+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(SCHEM:"cyclic AMP") -> r(MGI:S100a8)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11342660

3
value: IL-10 decreased the optimal time of mRNA expression induced by LPS from 24 to 8 h. Blocking experiments indicated that endogenous IL-10 contributes to gene induction by LPS. Cooperation between IL-10 and LPS was not due to altered mRNA stability but was dependent on de novo protein synthesis. The region of the promoter involved was localized to a 178-bp fragment flanking the transcription start site of the gene. This region was also responsible for the suppressive effects of IL-4 and IL-13. Forskolin, CTP-cAMP, and PGE(2) also enhanced LPS- and IFN-induced A8 mRNA,
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11342660

RAF7LvE7i4VTWFipz IjZVIiO7vLFmQoz7hYFLXRxWed0
has assertion: p(PFM:"Il1 Family") -> r(MGI:S100a8)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.051+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(PFM:"Il1 Family") -> r(MGI:S100a8)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11342660

4
value: Up-regulation of the A8 gene by LPS (17), IFN-, IL-1, and TNF in elicited Mac (18) and by LPS and IL-1 in microvascular endothelial cells (MEC; 19) suggested a proinflammatory role.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11342660

RAJ8Jy11yBL3xyhFqhYgnRWkbDtwiGw5ZUzozSB4TIdec
has assertion: a(CHEBI:troglitazone) -> p(HGNC:CDKN2C)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.055+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:troglitazone) -> p(HGNC:CDKN2C)
label: p(HGNC:RB1) -| (a(CHEBI:troglitazone) -> p(HGNC:CDKN2C))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11343236

5
value: HLF, which was deficient in the protein product of the retinoblastoma tumor-suppressor gene (pRb), responded most profoundly to troglitazone, showing an increased expression in not only p21, but also in p27 and in p18.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11343236

RAEkVvSEx84JIhWSNYwBUkszqv9HRTMt RtoKBWaUGDuQ
has assertion: a(CHEBI:troglitazone) -> r(HGNC:CDKN2C)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.062+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:troglitazone) -> r(HGNC:CDKN2C)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11343236

3
value: HLF, which was deficient in the protein product of the retinoblastoma tumor-suppressor gene (pRb), responded most profoundly to troglitazone, showing an increased expression in not only p21, but also in p27 and in p18.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11343236

RAJUypS5Bp8nQoTAuvE2vRZ4fE4OanjmtEEkkHmDRTzOc
has assertion: a(CHEBI:acetaldehyde) -> (kin(complex(NCH:"p85/p110 PI3Kinase Complex")) -> kin(p(HGNC:RPS6KB1)))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.063+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:acetaldehyde) -> (kin(complex(NCH:"p85/p110 PI3Kinase Complex")) -> kin(p(HGNC:RPS6KB1)))
label: kin(complex(NCH:"p85/p110 PI3Kinase Complex")) -> kin(p(HGNC:RPS6KB1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11343241

6
value: As expected, acetaldehyde-elicited ERK1/2 phosphorylation was inhibited by PD98059, a MEK inhibitor, but not by wortmannin, a PI3K inhibitor. On the other hand, both of these inhibitors partially inhibited phosphorylation of pp70(S6K) induced by acetaldehyde suggesting that its activation is ERK1/2- and PI3K-dependent.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11343241

RAGnAQWjoNEm z1Qb9UEEyM1RUgCO3YZNhAMu93ojhJ2I
has assertion: a(CHEBI:acetaldehyde) -> (kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK3)))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.109+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:acetaldehyde) -> (kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK3)))
label: kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK3))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11343241

7
value: These effects were completely prevented by calphostin C, a protein kinase C inhibitor.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11343241

RALY14T9S9KEOxyO1bMwVwG-w8dc39IJwttIP6a1d9n7k
has assertion: a(CHEBI:acetaldehyde) -> (kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:RPS6KB1)))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.111+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:acetaldehyde) -> (kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:RPS6KB1)))
label: kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:RPS6KB1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11343241

7
value: These effects were completely prevented by calphostin C, a protein kinase C inhibitor.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11343241

RAJSyAJzVRlLeMMrJ426lnxOX9aQFvpeUEiAN3GhbnX E
has assertion: kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.116+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11343241

6
value: These effects were completely prevented by calphostin C, a protein kinase C inhibitor.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11343241

RAPii-LjT 6IZTyX-NfjlxpreoQa9OeLbKvqG9uLHbvhQ
has assertion: kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK3))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.118+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK3))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11343241

6
value: These effects were completely prevented by calphostin C, a protein kinase C inhibitor.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11343241

RAEynivuCxOjVH6ml3tGVbf 59lGjRqv2hZYJ9gh85XP4
has assertion: a(CHEBI:lipopolysaccharide) -| r(RGD:Cyp4f4)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.122+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:lipopolysaccharide) -| r(RGD:Cyp4f4)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11343254

3
value: See Figure 1: Toxicogenomic patterns of gene expression. All changes correspond to the LPS, 6 hour timepoint and are greater than or equal to 2.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11343254

RAILwcz8Gcen2pZ6FzmxvIGY0aSP4lG4I3glHH0IQUIFI
has assertion: cat(p(MGI:Ptgs2)) -> sec(a(CHEBI:serotonin))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.128+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

cat(p(MGI:Ptgs2)) -> sec(a(CHEBI:serotonin))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11344169

5
value: Pleckstrin phosphorylation and serotonin secretion in human platelets stimulated with vWF are blocked by the cyclooxygenase inhibitor acetylsalicylic acid
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11344169

RAGWpYmmkdd55R0tob1rx4dBpocIzw k7nkwXychOLcO4
has assertion: a(CHEBI:glucocorticoid) -> cat(p(HGNC:HSD11B1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.131+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:glucocorticoid) -> cat(p(HGNC:HSD11B1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11344242

4
value: 11betaHSD1 activity was sensitively up-regulated by increasing concentrations of glucocorticoid
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11344242

RAPg7SDWaStntYszQ49qZH3ifG8NgZ9z7Oag-M8UsUdXY
has assertion: kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.148+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(PFH:"PRKC Family")) -> kin(p(HGNC:MAPK1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11350732

6
value: PLCG1-mediated production of diacylglycerol leads to activation of PKC, and this pathway plays an important role in mediating VEGF-induced activation of ERKs ERK activation mediates cytosolic PLA2-mediated PGI2 synthesis
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11350732

RAHwtj0BPBwZJMdj9LnY6LJgeCBER6K0lV CqbmIHtCIE
has assertion: p(HGNC:IFNA1) -> p(HGNC:STAT2,pmod(P))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.174+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:IFNA1) -> p(HGNC:STAT2,pmod(P))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11353767

4
value: IFNA induced activation of STAT1 and STAT2.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11353767

RAPOYG32tdlppttffDefnLeP7LLToaJ97ud39UHtFdkns
has assertion: kin(p(PFH:"AKT Family")) => p(HGNC:GSK3A,pmod(P,S,21))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.189+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(PFH:"AKT Family")) => p(HGNC:GSK3A,pmod(P,S,21))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11356119

5
value: PKB phosphorylated GSK3a at Ser-21
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11356119

RAL478fWIQf3ngtgFI9xt3kPM6YBHhR1x9DGCavEJdcFc
has assertion: a(CHEBI:clofibrate) -> r(MGI:Acox2)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.191+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:clofibrate) -> r(MGI:Acox2)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11356172

3
value: these proliferators also induce simultaneously the expression of genes encoding acyl-CoA oxidase, enoyl-CoA hydratase-hydroxyacyl-CoA dehydrogenase [multifunctional enzyme] and thiolase [3-ketoacyl-CoA thiolase]
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11356172

RAM4gJadVugBki3P5V4XYMFAViZKdcZLQ M7WOCt r8xk
has assertion: a(CHEBI:estradiol) -> r(HGNC:GHSR)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.199+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:estradiol) -> r(HGNC:GHSR)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11356716

3
value: The enhancing effect of Pit-1 was much lower for the GHS-R promoter than for 344 nucleotides of the human GH promoter, in agreement with the significantly lower expression levels of the GHS-R in somatotroph pituitary cells.Thyroid hormone and estrogen lead to a significant stimulation; glucocorticoids lead to a significant inhibition.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11356716

RAJ2IPFuv2DGFCiCaKSX0ilith16ahvus6Axg6T0DPnpI
has assertion: a(SCHEM:"LY 294002") -> bp(MESHPP:Apoptosis)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.224+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(SCHEM:"LY 294002") -> bp(MESHPP:Apoptosis)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11358816

2
value: After treating NSCLC cells containing active Akt/PKB with LY294002 for 48 h, typical morphological changes of apoptosis were observed, including membrane blebbing, increased refractoriness, and chromatin condensation (data not shown).
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11358816

RAIMiR79Am-HiJx91EBNRFFchw20uA2UNjZs5a1aj-y6Q
has assertion: p(HGNC:TGFB1) -| p(HGNC:KRT19)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.225+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:TGFB1) -| p(HGNC:KRT19)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11358848

4
value: TGF-beta1 stimulation of COLO-357, PANC-1, and IMIM-PC1 cells lead to a down-regulation of cytokeratins 8 and 19 mRNA levels in PANC-1 and IMIM-PC1 (responsive cell lines)cells and to a minor extent in COLO-357 cells. The mesenchymal differentiation marker vimentin and collagen1alpha is strongly up-regulated in all responsive cell lines. The TGF-beta1 mediated regulation of differentiation markers occurs in a time-dependent manner. In contrast, TGF-beta1 treatment failed to induce any significant change in the expression pattern of differentiation markers in cell lines with a nonfunctional TGF-beta1 signaling pathway (IMIM-PC2 and CAPAN-1). The TGF-beta1 mediated changes of gene expression were also reproducibly observed on the protein level in all responsive cell lines in multiple experiments; however, down-regulation of epithelial marker proteins was less prominent on the RNA level.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11358848

RAJtTTmD7CkYoCbdSBD4aaqvoGTgCJydtLSnPVy4JPrlU
has assertion: a(CHEBI:cisplatin) -| path(SDIS:"tumor growth")
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.228+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:cisplatin) -| path(SDIS:"tumor growth")
label: p(HGNC:DNAJC15) -> (a(CHEBI:cisplatin) -| path(SDIS:"tumor growth"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11358853

4
value: Loss of expression of a new member of the DNAJ protein family confers resistance to chemotherapeutic agents used in the treatment of ovarian cancer. MCJ expression was associated with enhanced sensitivity to paclitaxel, topotecan, and cisplatin, suggesting that MCJ loss may play a role in de novo chemoresistance in ovarian carcinoma.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11358853

RAH3XW990pSfgeCwHc3Thgby4QhzYEJlnO4qBPPDxYKGc
has assertion: kin(p(PFH:"PRKC Family")) -> cat(p(HGNC:FCGR3B))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.245+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(PFH:"PRKC Family")) -> cat(p(HGNC:FCGR3B))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11359830

6
value: Treatment of PMN with phosphatidylinositol-specific phospholipase C to remove FcgammaRIIIb also decreased phagocytosis of SAP-opsonized zymosan, but not CRP-opsonized zymosan.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11359830

RAPbqfjfu-XB8dBfkKqoiaV0ADfHryIQbdgCNk0LXIUqc
has assertion: complex(p(HGNC:APCS),p(HGNC:FCGR3B)) -> bp(GO:phagocytosis)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.247+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

complex(p(HGNC:APCS),p(HGNC:FCGR3B)) -> bp(GO:phagocytosis)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11359830

5
value: Zymosan was used to test the functional consequences of SAP and CRP binding to FcgammaR. Both SAP and CRP bound to zymosan and enhanced its uptake by PMN. This enhanced phagocytosis was abrogated by treatment of PMN with wortmannin, a phosphatidylinositol-3 kinase inhibitor, or with piceatannol, a Syk inhibitor, consistent with uptake through FcgammaR.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11359830

RAHekw7FizEhz3474thdmitPAEPPRmWHz sRREMXLZu8w
has assertion: p(HGNC:NGF) -> r(HGNC:APP)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.250+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:NGF) -> r(HGNC:APP)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11359873

4
value: These results demonstrate that NGF increases APP gene expression at transcriptional level.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11359873

RANxJzwTNuXnFiCAl0OmqHPHM 0tg0aS6MhydUgNqTQac
has assertion: p(HGNC:IGF1) -> kin(p(PFH:"AKT Family"))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.261+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:IGF1) -> kin(p(PFH:"AKT Family"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11359902

5
value: Activation of Akt is stimulated by a variety of growth factors, such as insulin, insulin-like growth factor 1 (IGF-1), platelet-derived growth factor, and epidermal growth factor
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11359902

RAEWD-pGwqneIjXKgK6BUYkvXIt0RTato z4 HKI8lPbA
has assertion: a(SCHEM:"serum insulin") -> kin(p(PFH:"AKT Family"))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.271+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(SCHEM:"serum insulin") -> kin(p(PFH:"AKT Family"))
label: phos(p(HGNC:PTEN)) -| (a(SCHEM:"serum insulin") -> kin(p(PFH:"AKT Family")))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11359902

7
value: PTEN has been shown to downregulate insulin, IGF-1, and epidermal growth factor- stimulated activation of Akt,
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11359902

RAJiC4BH9Sizd8QkhrnSdfUyY4DgRwnnlaaGNX0hvkH7w
has assertion: p(EGID:183) -> r(HGNC:CYP11B2)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.286+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(EGID:183) -> r(HGNC:CYP11B2)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11369439

4
value: Angiotensin-II (A-II) induces proliferation of zona glomerulosa cells and stimulates expression of cytochrome P-450 aldosterone synthase.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11369439

RAFBHoDIpDxopTkp8d5n-Xku47-i-uMybmZK2ochpNegw
has assertion: a(SCHEM:"AG-490") -| (p(HGNC:CXCR4) -> kin(complex(NCH:"p85/p110 PI3Kinase Complex")))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.287+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(SCHEM:"AG-490") -| (p(HGNC:CXCR4) -> kin(complex(NCH:"p85/p110 PI3Kinase Complex")))
label: p(HGNC:CXCR4) -> kin(complex(NCH:"p85/p110 PI3Kinase Complex"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11369622

5
value: Furthermore, pretreatment of CTS cells with AG490 significantly inhibited SDF-1alpha-induced PI3-kinase activity, and inhibition of JAK2 with AG490 ablated the SDF-1alpha-induced tyrosine phosphorylation of multiple focal adhesion proteins (including focal adhesion kinase, related adhesion focal tyrosine kinase, paxillin, CrkII, CrkL, and p130Cas).
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11369622

RAKrNJk59zzjSATkpPuVJy1VJ2fNUr9UfGL8UHs1kEoPs
has assertion: a(CHEBI:lipopolysaccharide) -> cat(p(MGI:Casp3))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.291+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:lipopolysaccharide) -> cat(p(MGI:Casp3))
label: p(MGI:Bid) -> (a(CHEBI:lipopolysaccharide) -> cat(p(MGI:Casp3)))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11369777

6
value: caspase 3 activities are much higher in treated wild-type mice than in treated bid-deficient mice. Wild-type (solid bar) or bid-deficient (open bar) C57BL/6J or 129/SvJ mice were treated with 2 ?g/kg TNF? or LPS (50 or 25 ?g/kg) in the presence of 20 mg of GalN per mouse for 4 or 6 h as indicated.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11369777

RAFwnMAqPyK5Btg4BUkpSd-9-DodVjiaIu6-wP5kURICE
has assertion: p(HGNC:LGALS8) -> kin(p(HGNC:PTK2))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.293+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:LGALS8) -> kin(p(HGNC:PTK2))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11371555

5
value: galectin-8 triggers integrin-mediated signaling cascades including Tyr phosphorylation of focal adhesion kinase and paxillin
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11371555

RAJOgFG82zoOzM7pnAqaYtTab0t0PQlAq79KEIwh8 Z4k
has assertion: bp(GO:"response to osmotic stress") -> r(RGD:Akr1b1)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.323+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

bp(GO:"response to osmotic stress") -> r(RGD:Akr1b1)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11374073

3
value: Like SMIT, BGT1 and AR genes, TauT, CDO and CSD genes appear to be tonicity-sensitive genes which can be activated in vivo by hypertonicity in the rat kidney
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11374073

RAPIfLA 3nfFAze gzp7tJshzQ1UtbasjTWmzcxsQE15c
has assertion: bp(GO:"response to osmotic stress") -> r(RGD:Slc5a3)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.328+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

bp(GO:"response to osmotic stress") -> r(RGD:Slc5a3)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11374073

3
value: Like SMIT, BGT1 and AR genes, TauT, CDO and CSD genes appear to be tonicity-sensitive genes which can be activated in vivo by hypertonicity in the rat kidney
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11374073

RAMGxSbW2e-wJClEGZA0nfCa0OOP668m iTXmGGDXl6dc
has assertion: gtp(p(RGD:Rac1)) -> cat(p(RGD:Alox5))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.338+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

gtp(p(RGD:Rac1)) -> cat(p(RGD:Alox5))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11374881

6
value: These effects were markedly attenuated in Rat2-Rac(N17) cells expressing a dominant negative Rac1 mutant. Pretreatment with SB203580, a specific inhibitor of p38 MAP kinase
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11374881

RAEY72cgTmyD 5h5k0UcbyqBReNXNHEEMl8B8YOD32WSU
has assertion: cat(p(HGNC:AGER)) -> kin(p(HGNC:MAPK1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.358+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

cat(p(HGNC:AGER)) -> kin(p(HGNC:MAPK1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11375353

6
value: CML-modified albumin produced rapid transient activation of tyrosine phosphorylation, extracellular signal-regulated kinase 1 and 2, and p38 mitogen-activated protein kinase (MAPK)
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11375353

RALnLBlnlB0k-NSuuGdEFh5Bsuk wyrfpmQpQayKqE es
has assertion: kin(p(HGNC:SRC)) => p(HGNC:CTNND1,pmod(P,Y,112))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.370+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(HGNC:SRC)) => p(HGNC:CTNND1,pmod(P,Y,112))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11382764

5
value: Here, as a first step toward pinpointing this role, we have employed two-dimensional tryptic mapping to directly identify the major sites of Src-induced p120 phosphorylation. Eight sites were identified by direct mutation of candidate tyrosines to phenylalanine and elimination of the accompanying spots on the two-dimensional maps. Identical sites were observed in vitro and in vivo, strongly suggesting that the physiologically important sites have been correctly identified. (Next section taken from Results - Fig 4) Mutation of tyrosines 112, 228, and 280 to phenylalanine resulted in elimination of phosphopeptide spots 6, 7, and 8, respectively. Mutation of tyrosine 96 eliminated spots 9 and 10, whereas mutation of tyrosine 257 eliminated spots 4 and 5...These results suggest that peptides 1, 2, and 3 represent singly, doubly, and triply phosphorylated variants, respectively, of the same peptide, containing Tyr291, Tyr296, and Tyr302.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11382764

RANL6W8npEe2ry9NWOjHjEFsM5Q5KzhWrqIxhO23uOIIY
has assertion: p(HGNC:BMX) -| bp(GO:"cell adhesion")
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.382+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:BMX) -| bp(GO:"cell adhesion")
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11382770

3
value: The expression of wild-type Etk in a non-invasive human breast cancer MCF-7 cells significantly increased proliferation and anchorage-independent growth of epithelial cancer cells.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11382770

RAJkmtsZaCkM YvGW0I6BMHVW7Dm10DVp20DLcibxV7cY
has assertion: p(HGNC:MYL9,pmod(P,T,18)) -> cat(p(HGNC:TNFRSF1B))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.389+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:MYL9,pmod(P,T,18)) -> cat(p(HGNC:TNFRSF1B))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11384975

5
value: Treatment of cells with either ionomycin or endotoxin (lipopolysaccharide) leads to activation of myosin II and increased translocation of TNFR-1 to the plasma membrane independent of TNF signaling.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11384975

RAGXMJzuIVHmuUq41L57Ar ecV716-P8 0ygSHqUOUf3g
has assertion: a(CHEBI:progesterone) -| r(HGNC:LEPR)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.392+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:progesterone) -| r(HGNC:LEPR)
label: p(HGNC:PGR) -> (a(CHEBI:progesterone) -| r(HGNC:LEPR))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11385112

5
value: These results indicate that, in the human endometrium, progestins act via the progesterone receptors to suppress functional OB-R(L) mRNA expression, and may thereby alter the sensitivity of the endometrium to leptin.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11385112

RAOQ 7S RcEPS2bK9DD593zRfBgAJlkU0-OjYVYIXHBms
has assertion: a(CHEBI:progesterone) -| r(HGNC:LEPR)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.395+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:progesterone) -| r(HGNC:LEPR)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11385112

3
value: whereas oestradiol plus progesterone (10(-8), 10(-7) and 10(-6) mol/l) or medroxyprogesterone acetate (10(-8) and 10(-7) mol/l) suppressed the expression by approximately 50%.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11385112

RAHg3r6cqU0IPtcwbYLR2IeH7I5gU2bHqc5EwN 12b0oI
has assertion: p(HGNC:GAS2) -> bp(MESHPP:Apoptosis)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.404+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:GAS2) -> bp(MESHPP:Apoptosis)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11387205

3
value: Gas2 is a caspase-3 substrate that plays a role in regulating microfilament and cell shape changes during apoptosis. Here we provide evidence that overexpression of Gas2 efficiently increases cell susceptibility to apoptosis following UV irradiation, etoposide and methyl methanesulfonate treatments, and that these effects are dependent on increased p53 stability and transcription activity.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11387205

RAP7zS2K5nl4zzoVpN8Ux8PHHftmfpDJ6Bfv2nGyAjx5M
has assertion: p(HGNC:TGFB1) -> complex(p(HGNC:DAB2),p(HGNC:SMAD3))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.413+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:TGFB1) -> complex(p(HGNC:DAB2),p(HGNC:SMAD3))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11387212

6
value: TGFbeta stimulation triggers a transient increase in association of Dab2 with Smad2 and Smad3
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11387212

RAEqjI8i4e7UDugNHQUAHaX3P4tgK9lgM-NrwnuPZfTJk
has assertion: p(HGNC:TGFB1) -> complex(p(HGNC:DAB2),p(HGNC:SMAD2))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.416+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:TGFB1) -> complex(p(HGNC:DAB2),p(HGNC:SMAD2))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11387212

6
value: TGFbeta stimulation triggers a transient increase in association of Dab2 with Smad2 and Smad3,
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11387212

RAHI0-WeKb5A3YsgzY6CCGuUhWJvr5z80WIrHP7y0rvRs
has assertion: p(HGNC:AKT3,pmod(P,T,305)) => kin(p(HGNC:AKT3))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.433+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:AKT3,pmod(P,T,305)) => kin(p(HGNC:AKT3))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11387345

5
value: When Thr-305 of HA-PKB?1 was mutated to an alanine that cannot be phosphorylated (HA-PKB?1T305A), the activation was abolished (Fig. 4 A).
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11387345

RAEFo-f06OxAYhCNn1znRPymz-NNb1PQsyRRS0UJJ4gG4
has assertion: p(MGI:Akt2) -| a(SCHEM:"D-Glucose, anhydrous")
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.435+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(MGI:Akt2) -| a(SCHEM:"D-Glucose, anhydrous")
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11387480

3
value: ablation of Akt2 in mice results in mild, but statistically significant fasting hyperglycemia due to peripheral insulin resistance and nonsuppressible hepatic glucose production accompanied by inadequate compensatory hyperinsulinemia
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11387480

RAIsUeiGPhZdWNecTFNy2tEOHZjPfGcoayYjc-uj3lp0s
has assertion: cat(p(HGNC:SIAH1)) -| p(HGNC:CTNNB1)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.441+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

cat(p(HGNC:SIAH1)) -| p(HGNC:CTNNB1)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11389840

5
value: Modified assertion
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11389840

RANFAW3t pU23PTnCeISKzU6PtJW1SZ3NCIbN5f-AXICg
has assertion: complex(p(HGNC:TNFAIP3),p(HGNC:TAX1BP1)) -| bp(MESHPP:Apoptosis)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.446+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

complex(p(HGNC:TNFAIP3),p(HGNC:TAX1BP1)) -| bp(MESHPP:Apoptosis)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11389905

5
value: A similar functional redundancy of the A20 zinc fingers was also observed for binding of A20 to a number of other proteins, including two novel NF-kappaB inhibitory proteins (ABIN-1, ABIN-2), A20 itself, the anti-apoptotic protein TXBP151, and a regulatory component of the IkappaB kinase complex, IKKgamma.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11389905

RAKV4e3k7Oq3tlwOMXAomuvK 8btUe8MpODiCvVjU0F6w
has assertion: p(MGI:Abl1) -> p(MGI:Plscr2)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.456+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(MGI:Abl1) -> p(MGI:Plscr2)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11390389

4
value: Phospholipid scramblase 1 (PLSCR1) is a plasma membrane protein that has been proposed to play a role in the transbilayer movement of plasma membrane phospholipids. PLSCR1 contains multiple proline-rich motifs resembling Src homology 3 (SH3) domain-binding sites, which highly specific for binding to the Abl SH3 domain. Endogenous PLSCR1 was immunoprecipitated from either Abl-/- or Abl+/+ murine fibroblasts using antibody specific for murine PLSCR1 the data indicated that Tyr phosphorylation of PLSCR1 detected in the Abl+/+ fibroblasts was virtually abolished in the Abl-/- fibroblasts, implying that the c-Abl kinase accounts for most, if not all, of the Tyr phosphorylation of PLSCR1 detected in mammalian cells.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11390389

RAFlJAMh4JhNW7IAcMQDE2RMCCx-ROffZdp5Twl 23PII
has assertion: cat(p(HGNC:GFPT1)) -| tscript(p(HGNC:PDX1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.459+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

cat(p(HGNC:GFPT1)) -| tscript(p(HGNC:PDX1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11390407

6
value: Overexpression of GFAT or treatment with glucosamine results in impaired glucose-stimulated insulin secretion and reduction in the expression levels of several beta-cell specific genes (insulin, GLUT2, and glucokinase). Additionally, the DNA binding activity of PDX-1, an important transcription factor for these three genes, was markedly reduced.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11390407

RAMZDx4EFK8kYziajPta-WY1yZTzwr8V900ej-xT4E3vc
has assertion: cat(p(HGNC:FRS2)) -> (kin(p(HGNC:RET)) -> kin(p(PFH:"MAPK Erk1/3 Family")))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.474+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

cat(p(HGNC:FRS2)) -> (kin(p(HGNC:RET)) -> kin(p(PFH:"MAPK Erk1/3 Family")))
label: kin(p(HGNC:RET)) -> kin(p(PFH:"MAPK Erk1/3 Family"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11390647

9
value: However, overexpression of FRS2 but not Shc potentiates mitogen-activated protein (MAP) kinase activation by RET oncoproteins.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11390647

RAGYM7Df5o wcbhQlSwECUXKDM5qzCR3itbzYWQmsp73s
has assertion: kin(p(HGNC:RET)) -> kin(p(PFH:"MAPK Erk1/3 Family"))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.476+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(HGNC:RET)) -> kin(p(PFH:"MAPK Erk1/3 Family"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11390647

6
value: However, overexpression of FRS2 but not Shc potentiates mitogen-activated protein (MAP) kinase activation by RET oncoproteins.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11390647

RAEoU3Qp- QNUsVo4nD9 Lvg2nYcaFMmQUouGFAnCJqug
has assertion: complex(NCH:"Catalytic Activity of T-cell receptor complex") -> kin(p(HGNC:MAPK3))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.484+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

complex(NCH:"Catalytic Activity of T-cell receptor complex") -> kin(p(HGNC:MAPK3))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11390650

4
value: The P-1 domain mediates a constitutive interaction of SLP-76 with the SH3 domain of PLC-gamma1 and is required for TCR-mediated activation of Erk, PLC-gamma1, and NFAT (nuclear factor of activated T cells).
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11390650

RAHaZw3nBpOSmOqKf7wG2NSndF8TrAmJTSEkiJ3rTGitA
has assertion: p(HGNC:EGF) -> p(HGNC:MCF2,pmod(P,Y))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.494+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:EGF) -> p(HGNC:MCF2,pmod(P,Y))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11394904

4
value: Here we describe that epidermal growth factor stimulates the ACK1/Dbl pathway, leading to actin cytoskeletal rearrangements. The role of the two ACK1-binding proteins Cdc42 and Grb2 was assessed by overexpression of the Cdc42/Rac interactive binding domain and a dominant-negative Grb2 mutant, respectively.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11394904

RAF27NN M-q0Knx3eFUYJmYJvxZllveqxaku UXAUw Xw
has assertion: p(HGNC:BMP2) -> cat(p(HGNC:CASP9))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.500+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:BMP2) -> cat(p(HGNC:CASP9))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11395480

5
value: Consistent with these results, BMP-2 increases caspase-9 and caspase-3, -6, and -7 activity, and an anti-caspase-9 agent suppresses BMP-2-induced apoptosis.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11395480

RAL sULgIv0D8AvpfnTQ-V80d azCRqpgqdHOmsQC3wNc
has assertion: a(CHEBI:glucocorticoid) -| r(HGNC:IL2)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.513+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:glucocorticoid) -| r(HGNC:IL2)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11397794

3
value: glucocorticoid-induced inhibition of interleukin-2 mRNA expression
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11397794

RAJJ0j5Dhm9exe 7UNSVycFd9bAec5i 42hqZXOnzT-JE
has assertion: cat(p(RGD:Adra2c)) -> kin(p(PFR:"Prkc Family"))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.518+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

cat(p(RGD:Adra2c)) -> kin(p(PFR:"Prkc Family"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11398898

6
value: The alpha2 adrenoceptor agonist, UK14,304-induced sustained contraction and Ca2+ oscillations may be due to PKC activation and opening
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11398898

RAOYKMxxiDzJj1rzGJA8X-OCFbZ9dIvS5O4TtYi0EmoUA
has assertion: p(HGNC:COL4A3) -| bp(GO:angiogenesis)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.520+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:COL4A3) -| bp(GO:angiogenesis)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11399763

3
value: Recently, we reported that tumstatin (the NC1 domain of alpha 3 chain of type IV collagen) and its deletion mutant tum-5 possess anti-angiogenic activity. In the present study, we confirm that the anti-angiogenic activity of tumstatin and tum-5 is independent of disulfide bond requirement.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11399763

RANcdSlFPC59qAr8hXvsj64nsI5WWd5dloYn6 bnLt5CA
has assertion: p(HGNC:FGF1) -> p(HGNC:CDH1,pmod(P,Y))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.523+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:FGF1) -> p(HGNC:CDH1,pmod(P,Y))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401320

4
value: Here we show that exposure to FGF-1 and FGF-2 leads to significant and dose-dependent increase in E-cadherin-dependent cell-cell adhesion, tubular differentiation, and a reduced capacity to invade collagen gels. FGF stimulation produces phosphorylation of E-cadherin and beta-catenin on tyrosine residues, as well as increased E-cadherin localisation to the cytoplasmic membrane and association with FGFR1 demonstrable by coimmunoprecipitation
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401320

RAK5VwgHYnm4YdtM0FXKvgZvdD0vioAKH01rNbhvaOhGU
has assertion: p(HGNC:FGF1) -> p(HGNC:CTNNB1,pmod(P,Y))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.525+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:FGF1) -> p(HGNC:CTNNB1,pmod(P,Y))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401320

4
value: Here we show that exposure to FGF-1 and FGF-2 leads to significant and dose-dependent increase in E-cadherin-dependent cell-cell adhesion, tubular differentiation, and a reduced capacity to invade collagen gels. FGF stimulation produces phosphorylation of E-cadherin and beta-catenin on tyrosine residues, as well as increased E-cadherin localisation to the cytoplasmic membrane and association with FGFR1 demonstrable by coimmunoprecipitation
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401320

RAFIvoyO 6ak8XoxQPbujz1seaQl86Ogfxg5b9jdovzQU
has assertion: p(HGNC:FGF1) -> complex(p(HGNC:CDH1),p(HGNC:FGFR1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.529+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:FGF1) -> complex(p(HGNC:CDH1),p(HGNC:FGFR1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401320

6
value: Here we show that exposure to FGF-1 and FGF-2 leads to significant and dose-dependent increase in E-cadherin-dependent cell-cell adhesion, tubular differentiation, and a reduced capacity to invade collagen gels. FGF stimulation produces phosphorylation of E-cadherin and beta-catenin on tyrosine residues, as well as increased E-cadherin localisation to the cytoplasmic membrane and association with FGFR1 demonstrable by coimmunoprecipitation
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401320

RAKnn4z ZJ-cSHG 65xCxZhPc2OxBC6XBSqOZxVYCpac8
has assertion: p(HGNC:FGF1) -> tscript(p(PFH:"TCF/LEF Family"))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.531+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:FGF1) -> tscript(p(PFH:"TCF/LEF Family"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401320

5
value: Here we show that exposure to FGF-1 and FGF-2 leads to significant and dose-dependent increase in E-cadherin-dependent cell-cell adhesion, tubular differentiation, and a reduced capacity to invade collagen gels. FGF stimulation produces phosphorylation of E-cadherin and beta-catenin on tyrosine residues, as well as increased E-cadherin localisation to the cytoplasmic membrane and association with FGFR1 demonstrable by coimmunoprecipitation
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401320

RAJrwFUgpsfhOuaZx2DHSq2KH4ZxsV hitO9R809oRa7M
has assertion: p(HGNC:FGF2) -> p(HGNC:CTNNB1,pmod(P,Y))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.532+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:FGF2) -> p(HGNC:CTNNB1,pmod(P,Y))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401320

4
value: Here we show that exposure to FGF-1 and FGF-2 leads to significant and dose-dependent increase in E-cadherin-dependent cell-cell adhesion, tubular differentiation, and a reduced capacity to invade collagen gels. FGF stimulation produces phosphorylation of E-cadherin and beta-catenin on tyrosine residues, as well as increased E-cadherin localisation to the cytoplasmic membrane and association with FGFR1 demonstrable by coimmunoprecipitation
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401320

RALa3G ulXKIo3YbYOqXRLoh6sUi99AyRxbvNnCQknnfQ
has assertion: p(HGNC:FGF2) -> complex(p(HGNC:CDH1),p(HGNC:FGFR1))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.535+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:FGF2) -> complex(p(HGNC:CDH1),p(HGNC:FGFR1))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401320

6
value: Here we show that exposure to FGF-1 and FGF-2 leads to significant and dose-dependent increase in E-cadherin-dependent cell-cell adhesion, tubular differentiation, and a reduced capacity to invade collagen gels. FGF stimulation produces phosphorylation of E-cadherin and beta-catenin on tyrosine residues, as well as increased E-cadherin localisation to the cytoplasmic membrane and association with FGFR1 demonstrable by coimmunoprecipitation
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401320

RAKtuKqS7sgu6M9smSANBGAZYnnTv zIi6LVF94WoQggw
has assertion: p(MGI:Bmp4) -> bp(GO:"cell differentiation")
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.540+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(MGI:Bmp4) -> bp(GO:"cell differentiation")
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401399

3
value: Recombinant CHL protein inhibited the BMP4-dependent differentiation of embryonic stem cells in vitro and interacted directly with BMPs, similar to chordin.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401399

RAN60SVDaQK0I lXQOJKBT1BL9SXuC1 u71DFJ4S8wEVA
has assertion: p(MGI:Bmp4) -> bp(GO:"cell differentiation")
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.541+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(MGI:Bmp4) -> bp(GO:"cell differentiation")
label: p(MGI:Chrdl1) -| (p(MGI:Bmp4) -> bp(GO:"cell differentiation"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401399

5
value: Recombinant CHL protein inhibited the BMP4-dependent differentiation of embryonic stem cells in vitro and interacted directly with BMPs, similar to chordin.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401399

RAPeCgrImsKHG9ftev7tj0Ncgi6AU G0YKMz VwVM1shE
has assertion: a(CHEBI:androgen) -> r(MGI:Tra2a)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.544+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:androgen) -> r(MGI:Tra2a)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11401487

3
value: Androgens upregulate the expression of tra-2alpha, an enhancer of RNA splicing homologous to Drosophila tra-2, and promote redistribution of the protein from a diffuse to a speckled pattern within the nucleus. Similarly, androgens upregulate the expression of a novel gene homologous to Caenorhabditis elegans fox-1
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11401487

RAPpSWgmVL0Or7kTpcSHCYYrZmkaW5Ift2i0AnFtYfMMQ
has assertion: p(HGNC:HSPD1) -> complex(p(HGNC:TLR4),p(HGNC:LY96))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.552+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:HSPD1) -> complex(p(HGNC:TLR4),p(HGNC:LY96))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11402040

6
value: Human fibroblasts transfected with TLR2 or TLR4 plus MD-2 gain responsiveness to HSP60, while TLR2- or TLR4-defective cells display impaired responses.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11402040

RANJ9MHEjQuQdjYKIzQ5ApR aTAS9UzpqBevAdbLJLPSU
has assertion: a(CHEBI:nicotine) -> p(MGI:Chrna7)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.572+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:nicotine) -> p(MGI:Chrna7)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11404397

3
value: Nicotine (500 microM) activated 42 kDa isoform of MAPK in hippocampi from L250T animals. Stimulation of ERK2 was dependent on alpha7 nAChR function because an alpha7-selective antagonist, methyllycaconitine (MLA, 1 microM), attenuated nicotine-induced activation of ERK2 MAPK in L250T hippocampal slices. These results suggested that nicotine induced the alpha7 nAChR activation in hippocampus.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11404397

RAGjoTB3B8pewc4 P2m8DcTnKSR1KVHNdozziuc9TU7SI
has assertion: p(PFH:"AKT Family") -| tscript(p(HGNC:AR))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.575+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(PFH:"AKT Family") -| tscript(p(HGNC:AR))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11404460

5
value: A mutation at AR Ser-210 results in the reversal of Akt-mediated suppression of AR transactivation.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11404460

RAGHLnDwdOqZPjFDFdDY0FuQRcCx6j-IXByHa7tfqDLBc
has assertion: kin(p(HGNC:AKT1)) => p(HGNC:AR,pmod(P,S,210))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.580+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(HGNC:AKT1)) => p(HGNC:AR,pmod(P,S,210))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11404460

5
value: Here, we demonstrate that Akt phosphorylates the androgen receptor (AR) at Ser-210 and Ser-790.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11404460

RAJN5DJUZpHseEBvgdmEc01j2HU4cNyDUp5TCBR5mP1Eo
has assertion: p(HGNC:AR,pmod(P,S,790)) =| tscript(p(HGNC:AR))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.585+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:AR,pmod(P,S,790)) =| tscript(p(HGNC:AR))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11404460

5
value: we demonstrate that Akt phosphorylates the androgen receptor (AR) at Ser-210 and Ser-790.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11404460

RAGMCITNEsyrlZ1DrI9bokGT3qLV3XuEqpSHs5nrK3YNI
has assertion: p(HGNC:HGF) -> p(HGNC:PLAU)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.651+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:HGF) -> p(HGNC:PLAU)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11408346

4
value: HGF induces the expression of uPA. When the HGF antibody or the tyrosine kinase inhibitor was introduced the uPA expression was decreased.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11408346

RAIy5r-qfmCVvZmeauWacINTLlv TuE-ZAIcNQSXFIQD4
has assertion: a(CHEBI:atorvastatin) -> r(HGNC:NOS3)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.656+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:atorvastatin) -> r(HGNC:NOS3)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11408394

3
value: Moreover, atorvastatin caused an upregulation of ecNOS mRNA expression (138+/-7% of control) and an enhanced ecNOS activity in the vessel wall (209+/-46% of control).
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11408394

RAGyEhvfM1Ghz-IJniWol4u4C4ahLI PvORVcA8c6cc3E
has assertion: p(RGD:Ngf) -> p(RGD:Ntrk1)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.659+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(RGD:Ngf) -> p(RGD:Ntrk1)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11408601

4
value: PC-12 cells over expressing SH2-Bbeta when treated with 100 ng/ml of NGF for 10 minutes and immunoblotted with TrkA anti-phosphotyrosine antibody, showed normal NGF induced tyrosine phosphorylation of TrkA.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11408601

RAPHmk5ztqv2KtvdGl04PQIzU2boHf8vw TCkAjStqg 8
has assertion: p(HGNC:IL6) -> kin(p(HGNC:MAPK14))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.677+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:IL6) -> kin(p(HGNC:MAPK14))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11410519

5
value: Pretreatment of LNCaP cells with SB202190, PD98059, or tyrphostin AG879 [p38 mitogen-activated protein kinase (MAPK), MAP/extracellular signal-regulated protein kinase kinase 1/2, and ErbB2 MAPK inhibitors, respectively) but not wortmannin (PI3-kinase inhibitor) blocked IL-6-mediated induction of the PSA promoter, which demonstrates that IL-6 activity is dependent on a MAPK pathway.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11410519

RAMUOGCcpJqmGaS5S3Wd8ZzKHxwm0XVXrcMzOc3VspKtM
has assertion: p(HGNC:IKBKB) -> complex(NCH:"Nfkb Complex")
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.678+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:IKBKB) -> complex(NCH:"Nfkb Complex")
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11410591

3
value: IKK-beta brought about the activation of NFkB.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11410591

RAIWh3dbjI9JITu7pDmykMZoW5wxqo5SGpaoN1fibSL5U
has assertion: a(CHEBI:fulvestrant) -| r(RGD:Cuzd1)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.684+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:fulvestrant) -| r(RGD:Cuzd1)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11416020

3
value: # Ariadne: UO-44 gene expression in the uterus is strictly regulated by estrogens, tamoxifen, and GH, whereas the pure antiestrogen ICI 182780 is inhibitory. [Expression]
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11416020

RAGtrvr-QLKGO204QY6EQ7bs8O1KYtm5yHJgegP0WP DA
has assertion: p(HGNC:TNFSF10) -> p(HGNC:FADD)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.711+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:TNFSF10) -> p(HGNC:FADD)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11420695

4
value: PC-3 cells overexpressing FADD dominant negative form were found to be resistant to cell death induced by TRAIL.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11420695

RAFDG2b8OVeEAaEZEEwDigvc9KCTkNzRxiMWxNUeZa498
has assertion: p(HGNC:RAF1) -| bp(MESHPP:Apoptosis)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.734+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:RAF1) -| bp(MESHPP:Apoptosis)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11427728

3
value: Thus, Raf-1 may promote cell survival through its protein-protein interactions in addition to its established MEK kinase function
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11427728

RAHCNSFwrCribN5qkP6z Y vAD0FrKdzXWiUE8UWAKCA8
has assertion: a(CHEBI:androgen) -> (tscript(p(HGNC:AR)) -> tscript(p(HGNC:CREBBP)))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.740+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

a(CHEBI:androgen) -> (tscript(p(HGNC:AR)) -> tscript(p(HGNC:CREBBP)))
label: tscript(p(HGNC:AR)) -> tscript(p(HGNC:CREBBP))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429409

7
value: AR dramatically increased the intrinsic transcriptional activity of the nuclear receptor coactivators glucocorticoid receptor-interacting protein-1 (GRIP1), cAMP response element-binding protein-binding protein, and p300 that are tethered to DNA.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429409

RAGAuoB7YOs0yDiejndeoaqvwvygigKxZeVZt6reY8nxg
has assertion: p(HGNC:AR) -| tscript(complex(NCH:"Nfkb Complex"))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.752+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:AR) -| tscript(complex(NCH:"Nfkb Complex"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429409

4
value: In addition, liganded AR can interfere with the activity of other transcription factors, such as activator protein-1 and nuclear factor kappaB, for which DNA binding by AR is not necessary.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429409

RAL9vYNNl00-aObvA63eZI4oevQNG3QwN0hOBWyafsqoA
has assertion: p(HGNC:TP53,sub(F,134,I)) =| tscript(p(HGNC:TP53))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.755+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

1
has substitution: F,134,I
variant of: http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11998
type: http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000

p(HGNC:TP53,sub(F,134,I)) =| tscript(p(HGNC:TP53))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429705

5
value: Table 2 p53 mutants unable to discriminate between diÂ?erent p53 responsive elements
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429705

RAFCbAHKpRMlnw-hPLqP gEts390cU-7GJkH5fAZWN-qA
has assertion: p(HGNC:TP53,sub(Y,205,N)) =| tscript(p(HGNC:TP53))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.771+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

1
has substitution: Y,205,N
variant of: http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11998
type: http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000

p(HGNC:TP53,sub(Y,205,N)) =| tscript(p(HGNC:TP53))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429705

5
value: Table 2 p53 mutants unable to discriminate between diÂ?erent p53 responsive elements
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429705

RAO72uyXdD6fBOwdORw-TOKqxS2cw2PtXzSWG-H5fIT-s
has assertion: p(HGNC:TP53,sub(A,276,G)) =| tscript(p(HGNC:TP53))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.783+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

1
has substitution: A,276,G
variant of: http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11998
type: http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000

p(HGNC:TP53,sub(A,276,G)) =| tscript(p(HGNC:TP53))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429705

5
value: Table 2 p53 mutants unable to discriminate between diÃ???erent p53 responsive elements
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429705

RAJLjWIg618uW9FbXbnb8cO0Pd86GTsUFKxT4Z7vTGz1Q
has assertion: p(HGNC:TP53,sub(E,286,K)) =| tscript(p(HGNC:TP53))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.794+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

1
has substitution: E,286,K
variant of: http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11998
type: http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000

p(HGNC:TP53,sub(E,286,K)) =| tscript(p(HGNC:TP53))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429705

5
value: Table 2 p53 mutants unable to discriminate between diÃ???erent p53 responsive elements
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429705

RAHx0-0rUTf6VHY7s240fnbPCNiRpDa5zhATe8PM1zoRw
has assertion: p(HGNC:TP53,sub(G,244,S)) =| tscript(p(HGNC:TP53))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.816+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

1
has substitution: G,244,S
variant of: http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11998
type: http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000

p(HGNC:TP53,sub(G,244,S)) =| tscript(p(HGNC:TP53))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429705

5
value: Table 2 p53 mutants unable to discriminate between diÃ??erent p53 responsive elements
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429705

RAJGX-m3RdbvisddhV1lqyZfUP9n67hxAfSAVNS4CROa4
has assertion: p(HGNC:TP53,sub(N,239,S)) =| tscript(p(HGNC:TP53))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.828+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

1
has substitution: N,239,S
variant of: http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11998
type: http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000

p(HGNC:TP53,sub(N,239,S)) =| tscript(p(HGNC:TP53))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429705

5
value: Table 2 p53 mutants unable to discriminate between diÃ??erent p53 responsive elements
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429705

RAMlQO7GZRcoZOSjE9grclz5uMCLRqag4yZiEla TnG8A
has assertion: p(HGNC:TP53,sub(S,241,T)) =| tscript(p(HGNC:TP53))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.834+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

1
has substitution: S,241,T
variant of: http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11998
type: http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000

p(HGNC:TP53,sub(S,241,T)) =| tscript(p(HGNC:TP53))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429705

5
value: Table 2 p53 mutants unable to discriminate between diÃ??erent p53 responsive elements
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429705

RAMAU9xMT1aKN9YyC6VmnqHktQ6fMJDnqdsIeWRkR ZJE
has assertion: p(HGNC:TP53,sub(Y,205,N)) =| tscript(p(HGNC:TP53))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.860+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

1
has substitution: Y,205,N
variant of: http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11998
type: http://www.ebi.ac.uk/ontology-lookup/?termId=MOD:00000

p(HGNC:TP53,sub(Y,205,N)) =| tscript(p(HGNC:TP53))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11429705

5
value: Table 3 EÂ?ect of temperature on the ability of p53 mutants to discriminate between diÂ?erent p53 responsive elements
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11429705

RAML AtTki47Imi9BnknH8JsUPUVih8eoW2O-EUE4oKts
has assertion: kin(p(HGNC:STK11)) => p(HGNC:STK11,pmod(P,T,189))
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.869+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

kin(p(HGNC:STK11)) => p(HGNC:STK11,pmod(P,T,189))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11430832

5
value: PhosphoElm data from PMID 15212693
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11430832

RAK-oC oMj1M5qGa79P-guuhrPXkQgbkp03jeVuRKnY7c
has assertion: cat(p(HGNC:F2R)) -> tloc(p(EGID:200576),GOCCACC:"GO:0005737",GOCCACC:"GO:0005886")
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.889+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

cat(p(HGNC:F2R)) -> tloc(p(EGID:200576),GOCCACC:"GO:0005737",GOCCACC:"GO:0005886")
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11431481

6
value: After stimulation of the thrombin receptor, PAR1, or overexpression of a constitutively active variant of G alpha(q), PIP5K I alpha translocated to the plasma membrane. Movement of PIP5K I alpha to the cell membrane was dependent on both GTP-bound Rac and Rho
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11431481

RAE1NGvl9JP95zJqaLdyDce7Y8X04RkelTjE8iERm9oDc
has assertion: p(HGNC:HBEGF) -> bp(GO:"cell migration")
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.915+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:HBEGF) -> bp(GO:"cell migration")
label: p(HGNC:NRD1) -> (p(HGNC:HBEGF) -> bp(GO:"cell migration"))
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11432822

5
value: When overexpressed in cells, NRDc enhanced their migration in response to HB-EGF but not to EGF
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11432822

RAGXrkcZB8gNhTTJFAr1ldIVKCoXdVAfhjsGvOtfVv7tw
has assertion: p(HGNC:NOTCH1) -> r(HGNC:CDKN1A)
has provenance: provenance
has publication info: pubinfo
type: Nanopublication
created: 2014-07-03T14:29:56.923+02:00
created by: 0000-0001-6818-334X
created by: http://orcid.org/0000-0002-1267-0234

p(HGNC:NOTCH1) -> r(HGNC:CDKN1A)
had primary source: http://www.ncbi.nlm.nih.gov/pubmed/11432830

4
value: The RBP-Jkappa protein binds directly to the endogenous p21 promoter and p21 expression is induced specifically by activated Notch1 through RBP-Jkappa-dependent transcription.
was quoted from: http://www.ncbi.nlm.nih.gov/pubmed/11432830