@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix chebi: . @prefix RNA: . @prefix mgi: . @prefix geneProductOf: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:1096365; a RNA: . sub:_2 occursIn: species:10090; rdf:object sub:_1; rdf:predicate belv:increases; rdf:subject chebi:6494; a rdf:Statement . sub:assertion rdfs:label "a(CHEBI:lipopolysaccharide) -> r(MGI:Psme2)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "1.4" . sub:_3 prov:value "In livers of LPS-treated mice, we observed coordinate induction of genes whose products act in concert to present endogenous antigens (Fig. 4). This wave of increased expression, sustained between 3 and 12 h after LPS administration, included genes for the specialized proteasome components LMP2 (psmb9), LMP7 (psmb8), LMP10 (psmb10), PA28 (psme1), and PA28 (psme2); the peptide transporter components Tap1 (abcb2) and Tap2 (abcb3); the accessory protein tapasin (tapbp); and MHC class II proteins (H2-k, H2-d; Fig. 4 A and B). Transcripts for the constitutive proteasome -subunit 2 (psma2) were also increased, albeit slightly (Fig. 4 A and B)."; prov:wasQuotedFrom pubmed:12540827 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12540827; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:09.543+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }