@prefix dcterms: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix Protein: .
@prefix hgnc: .
@prefix geneProductOf: .
@prefix go: .
@prefix mesh: .
@prefix occursIn: .
@prefix species: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 geneProductOf: hgnc:1697;
a Protein: .
sub:_2 occursIn: mesh:D005347, mesh:D011471, species:9606;
rdf:object go:0008283;
rdf:predicate belv:increases;
rdf:subject sub:_1;
a rdf:Statement .
sub:assertion rdfs:label "p(HGNC:CD74) -> bp(GO:\"cell proliferation\")" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_4;
pav:version "1.4" .
sub:_3 prov:value "In a recent study by Meyer-Siegler et al. [52] , MIF influenced cell viability and invasiveness. In prostate cancer cells, androgen-independent prostate cancer cells required MIF-activated signal transduction pathways for both growth and invasion, which was in contrast to androgen- dependent cells. They demonstrated that the MIF cell surface receptor CD74 was only detected in androgen- independent tumor cells. Treatments directed against either CD74 or MIF resulted in decreased cell proliferation, MIF secretion and invasion.";
prov:wasQuotedFrom pubmed:18791328 .
sub:_4 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:18791328;
prov:wasDerivedFrom beldoc:, sub:_3 .
}
sub:pubinfo {
this: dcterms:created "2014-07-03T14:30:53.567+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}