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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAxud5qoMzSLbBRPnMWO7vPr2vLtFgNnPN9rn-eDPd19U#_3 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAxud5qoMzSLbBRPnMWO7vPr2vLtFgNnPN9rn-eDPd19U#_2 http://www.w3.org/ns/prov#value Skeletal muscle contains myofibers that differ greatly in their oxidative capacity. Prolonged electrical stimulation or exercise training can lead to a conversion of type II (fast-twitch) muscle fibers to type I (slow-twitch) fibers (1). This conversion is characterized by dramatic changes in the expression of a large number of genes that increase mitochondrial biogenesis, oxidative capacity, and amounts of a distinct set of contractile proteins characteristic of this fiber type (2). Several key factors in this signaling cascade have been identified recently. 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