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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://resource.belframework.org/belframework/20131211/namespace/selventa-named-complexes/Nfkb%20Complex
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0043234
http://www.tkuhn.ch/bel2nanopub/RAxSURiMKpW3IzJMLq1GMz_FvASZrrpuoiHdtndyZBMe4#_1
http://semanticscience.org/resource/SIO_000139
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http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=5013
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_33697
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tscript(complex(SCOMP:"Nfkb Complex")) => r(HGNC:HMOX1)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Electrophoretic mobility-shift assays of nuclear protein extracts from heme-treated and control cells with specific oligonucleotide probes containing binding sites for known transcription factors, including AP-1, AP-2, Sp1, NF-kappa B, CTF/NF1, TFIID, OKT1, and CREB, and oligonucleotides containing serum-, metal-, and glucocorticoid-responsive elements demonstrated a specific and marked increase in the NF-kappa B and AP-2 transcription factors and, to a lesser extent, an increase in AP-1. No significant increase in other transcription factors over the control, untreated cells was observed. DNase I footprint assays using purified transcription factors revealed the presence of NF-kappa B and AP-2 binding sites in the proximal part of the promoter region of the human HO-1 gene.
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http://www.ncbi.nlm.nih.gov/pubmed/8016102
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Selventa
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2014-07-03T14:33:48.220+02:00
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