@prefix this: <http://www.tkuhn.ch/bel2nanopub/RAx8DfN1AVGcjhdGAxoSLtrphIg6cp71mtp8BCMJQZcI8> .
@prefix sub: <http://www.tkuhn.ch/bel2nanopub/RAx8DfN1AVGcjhdGAxoSLtrphIg6cp71mtp8BCMJQZcI8#> .
@prefix beldoc: <http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix rdf: <http://www.w3.org/1999/02/22-rdf-syntax-ns#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix dct: <http://purl.org/dc/terms/> .
@prefix dce: <http://purl.org/dc/elements/1.1/> .
@prefix pav: <http://purl.org/pav/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix belv: <http://www.selventa.com/vocabulary/> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix chebi: <http://www.ebi.ac.uk/chebi/searchId.do?chebiId=> .
@prefix sdis: <http://resource.belframework.org/belframework/20131211/namespace/selventa-legacy-diseases/> .
@prefix obo: <http://purl.obolibrary.org/obo/> .
@prefix occursIn: <http://purl.obolibrary.org/obo/BFO_0000066> .
@prefix species: <http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=> .
@prefix pubmed: <http://www.ncbi.nlm.nih.gov/pubmed/> .
@prefix orcid: <http://orcid.org/> .
sub:Head {
   this: np:hasAssertion sub:assertion ;
    np:hasProvenance sub:provenance ;
    np:hasPublicationInfo sub:pubinfo ;
    a np:Nanopublication .
}
sub:assertion {
   sub:_1 occursIn: obo:CLO_0009465 , obo:CL_0000115 , obo:UBERON_0004535 , species:9606 ;
    rdf:object sdis:monocyte%20adherence ;
    rdf:predicate belv:decreases ;
    rdf:subject chebi:7583 ;
    a rdf:Statement .
  sub:assertion rdfs:label "a(CHEBI:\"nitric oxide\") -| path(SDIS:\"monocyte adherence\")" .
}
sub:provenance {
   beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_3 ;
    pav:version "20131211" .
  sub:_2 prov:value "Interactions between monocytes and endothelial cells are enhanced by endothelial cell senescence (89), thereby promoting atherogenesis. This change appears to be mediated by the upregulation of adhesion molecules and pro-inflammatory cytokines, as well as decreased production of NO by senescent endothelial cells (14, 85)." ;
    prov:wasQuotedFrom pubmed:17981770 .
  sub:_3 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:17981770 ;
    prov:wasDerivedFrom beldoc: , sub:_2 .
}
sub:pubinfo {
   this: dct:created "2014-07-03T14:33:13.502+02:00"^^xsd:dateTime ;
    pav:createdBy orcid:0000-0001-6818-334X , orcid:0000-0002-1267-0234 .
}