@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix prov: . @prefix obo: . @prefix Protein: . @prefix mgi: . @prefix geneProductOf: . @prefix hasPart: . @prefix ProteinComplex: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 hasPart: mgi:104737, sub:_2; occursIn: obo:EFO_0004037, species:10090; a ProteinComplex: . sub:_2 geneProductOf: mgi:1344313; a Protein: . sub:assertion rdfs:label "complex(p(MGI:Zeb1),g(MGI:Cdkn2b))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value "Next, we asked whether Zeb1 acts directly on the p21Cdkn1a and p15Ink4b gene promoters. In ChIP assays, Zeb1 was bound to both promoters, but no interaction was seen with the control Gapdh promoter (Fig. 8C-E). These results are consistent with the notion that Zeb1 is represses these CDKIs directly by binding to their promoters in vivo."; prov:wasQuotedFrom pubmed:18192284 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:18192284; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:14.460+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }