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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAwJyO_wyYLa5D_HTLu0WWRFvcxB6oQeBhE9nEijHTWm0#_5 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAwJyO_wyYLa5D_HTLu0WWRFvcxB6oQeBhE9nEijHTWm0#_4 http://www.w3.org/ns/prov#value The inhibition of this oncogenic cooperation re-induces the expression of the death receptor and restores the sensitivity to FAS-L-mediated apoptosis [27,50]. In addition to its interaction with JUN, STAT3 cooperates with the noncanonical nuclear factor-kB (NF-kB) signaling pathway to regulate apoptosis [51]. STAT3 facilitates the processing of the p52 subunit from its p100 precursor and interacts with p52 on DNA. 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