sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_5 ;
    pav:version "1.4" .
  sub:_4 prov:value "LDL(-) stimulation led to a modest 3-fold Ppara ligand binding domain activation; in the presence of LPL and LDL(-), PPARa activation increased 30-fold....This LPL treatment may have produced greater amounts of the ligands already present in LDL(–), converted less potent ligands to more potent forms, or both....To identify this PPAR?-activating component in LDL(–), hydrolytic products of LPL-treated LDL(–) were extracted and separated using HPLC-MS. The major lipophylic compounds in LDL(–), either with or without LPL treatment, were HpODE and HODE acids" ;
    prov:wasQuotedFrom pubmed:12878589 .
  sub:_5 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:12878589 ;
    prov:wasDerivedFrom beldoc: , sub:_4 .
}