@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:5360; a Protein: . sub:_2 geneProductOf: hgnc:1787; a Protein: . sub:_3 occursIn: species:9606; rdf:object sub:_2; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:ID1) -| p(HGNC:CDKN2A)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "p16INK4a was much lower or undetectable in all the Id-1 expressing clones of LNCaP cells, while approximately 2 to 3 fold increases in p16INK4a levels was observed in the controls and in the Id-1 negative clones, suggesting that expression of Id-1 reduced the p16INK4a protein levels in LNCaP cells."; prov:wasQuotedFrom pubmed:12016143 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12016143; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:31:49.199+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }