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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAu6zTHlm4aPnRXuj0LBCbEyBeeCPX46GkxfwD33e-FmQ#_4 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAu6zTHlm4aPnRXuj0LBCbEyBeeCPX46GkxfwD33e-FmQ#_3 http://www.w3.org/ns/prov#value In general, proinflammatory cytokines and TLR ligands suppressed mCMKLR1 expression (Fig. 5, upper panel). We observed the following hierarchy in mCMKLR1 suppression by TLR ligands: LPS O polyI:C O CpG. The proinflammatory cytokines IFN-g and TNF-a caused modest mCMKLR1 suppression as well, which was enhanced by coincubation with LPS. 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