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http://www.tkuhn.ch/bel2nanopub/RAspuNr47bEpSl_M1v1G0fDQzp5asmutavHsUnl59-EKM#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=11892
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p(HGNC:TNF) -> bp(GOBP:"regulation of vascular permeability")
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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possibly through extracellular signal-regulated kinase (ERK)-catalyzed caldesmon phosphorylation and subsequent alteration of actomyosin cross bridging. Likewise, the cytokine TNF- through its receptor (TNF-R) appears to mediate cytoskeletal rearrangement and eventual barrier dysfunction through a PKC-dependent pathway. Although TNF- produces an increase in MLC phosphorylation, this effect does not appear to contribute to the subsequent increase in EC permeability.
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Selventa
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2014-07-03T14:31:43.721+02:00
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