@prefix this: <http://rdf.disgenet.org/nanopublications.trig#NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix sio: <http://semanticscience.org/resource/> .
@prefix ncit: <http://ncicb.nci.nih.gov/xml/owl/EVS/Thesaurus.owl#> .
@prefix lld: <http://linkedlifedata.com/resource/umls/id/> .
@prefix miriam-gene: <http://identifiers.org/ncbigene/> .
@prefix miriam-pubmed: <http://identifiers.org/pubmed/> .
@prefix eco: <http://purl.obolibrary.org/obo/eco.owl#> .
@prefix wi: <http://purl.org/ontology/wi/core#> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix pav: <http://purl.org/pav/2.0/> .
@prefix prv: <http://purl.org/net/provenance/ns#> .
@prefix dcterms: <http://purl.org/dc/terms/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix dgn-np: <http://rdf.disgenet.org/nanopublications.trig#> .
@prefix dgn-gda: <http://rdf.disgenet.org/gene-disease-association.ttl#> .
@prefix dgn-void: <http://rdf.disgenet.org/v2.1.0/void.ttl#> .
dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_head {
  this: np:hasAssertion dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_assertion ;
    np:hasProvenance dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_provenance ;
    np:hasPublicationInfo dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_publicationInfo ;
    a np:Nanopublication .
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  dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_provenance a np:Provenance .
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}
dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_assertion {
  miriam-gene:3359 a ncit:C16612 .
  lld:C0022104 a ncit:C7057 .
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    a sio:SIO_001121 .
}
dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_provenance {
  dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_assertion dcterms:description "[The second objective is to review pharmacogenetics in IBS, with the focus on cytochrome P-450 metabolism of drugs used in IBS, modulation of motor and sensory responses to serotonergic agents based on the 5-hydroxytryptamine (5-HT) transporter-linked polymorphic region (5-HTTLPR) and 5-HT(3) genetic variants, responses to a nonselective cannabinoid agonist (dronabinol) based on cannabinoid receptor (CNR1) and fatty acid amide hydrolase (FAAH) variation, and responses to a bile acid (sodium chenodeoxycholate) and bile acid binding (colesevelam) based on klothoβ (KLB) and fibroblast growth factor receptor 4 (FGFR4) variation.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
    wi:evidence dgn-void:source_evidence_literature ;
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  dgn-void:source_evidence_literature a eco:ECO_0000212 ;
    rdfs:comment "Gene-disease associations inferred from text-mining the literature."@en ;
    rdfs:label "DisGeNET evidence - LITERATURE"@en .
}
dgn-np:NP828600.RArsd2nb_1-yssWHuemUPcvf3aKOXm0OndqwD9AqwWse4130_publicationInfo {
  this: dcterms:created "2014-10-02T12:40:27+02:00"^^xsd:dateTime ;
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    dcterms:rightsHolder dgn-void:IBIGroup ;
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