@prefix this: <http://rdf.disgenet.org/nanopublications.trig#NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix sio: <http://semanticscience.org/resource/> .
@prefix ncit: <http://ncicb.nci.nih.gov/xml/owl/EVS/Thesaurus.owl#> .
@prefix lld: <http://linkedlifedata.com/resource/umls/id/> .
@prefix miriam-gene: <http://identifiers.org/ncbigene/> .
@prefix miriam-pubmed: <http://identifiers.org/pubmed/> .
@prefix eco: <http://purl.obolibrary.org/obo/eco.owl#> .
@prefix wi: <http://purl.org/ontology/wi/core#> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix pav: <http://purl.org/pav/2.0/> .
@prefix prv: <http://purl.org/net/provenance/ns#> .
@prefix dcterms: <http://purl.org/dc/terms/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix dgn-np: <http://rdf.disgenet.org/nanopublications.trig#> .
@prefix dgn-gda: <http://rdf.disgenet.org/gene-disease-association.ttl#> .
@prefix dgn-void: <http://rdf.disgenet.org/v2.1.0/void.ttl#> .
dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_head {
  this: np:hasAssertion dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_assertion ;
    np:hasProvenance dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_provenance ;
    np:hasPublicationInfo dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_publicationInfo ;
    a np:Nanopublication .
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  dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_provenance a np:Provenance .
  dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_publicationInfo a np:PublicationInfo .
}
dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_assertion {
  miriam-gene:3091 a ncit:C16612 .
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}
dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_provenance {
  dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_assertion dcterms:description "[Sulfonamide derivative compounds with CA9 inhibitory characteristics represent one of the optimal treatment options beside other CA9 inhibitory agents or chemical inhibitory compounds against its main regulating transcription factor which is the hypoxia induced HIF-1α when applied against human cancers with hypoxic regions like GBM, bearing potential for an effective role in human brain tumour therapeutic strategies.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
    wi:evidence dgn-void:source_evidence_literature ;
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    prov:wasDerivedFrom dgn-void:befree-20140225 ;
    prov:wasGeneratedBy eco:ECO_0000203 .
  dgn-void:befree-20140225 pav:importedOn "2014-02-25"^^xsd:date .
  dgn-void:source_evidence_literature a eco:ECO_0000212 ;
    rdfs:comment "Gene-disease associations inferred from text-mining the literature."@en ;
    rdfs:label "DisGeNET evidence - LITERATURE"@en .
}
dgn-np:NP853002.RArhE117o7SujkNwzMoSb7Zhy1Hxmgd8WLfYUAZWe_ZTA130_publicationInfo {
  this: dcterms:created "2014-10-02T12:40:42+02:00"^^xsd:dateTime ;
    dcterms:rights <http://opendatacommons.org/licenses/odbl/1.0/> ;
    dcterms:rightsHolder dgn-void:IBIGroup ;
    dcterms:subject sio:SIO_000983 ;
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}