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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RArTDhec70PyjgLrkPGWK3JqGxEYLfDF0DP19JpbQZbzo#_1
http://semanticscience.org/resource/SIO_000139
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http://www.tkuhn.ch/bel2nanopub/RArTDhec70PyjgLrkPGWK3JqGxEYLfDF0DP19JpbQZbzo#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://amigo.geneontology.org/amigo/term/GO:0003824
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http://purl.obolibrary.org/obo/RO_0002204
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http://purl.obolibrary.org/obo/BFO_0000066
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path(SDIS:"viral infection") -> cat(p(HGNC:DDX58))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Polyinosinic-polycytidylic acid activated IFN regulatory factor 3 dimerization and phosphorylation, increased activity of the IFN-stimulated response element, induced a significant increase in IFN-beta mRNA transcripts and IFN-beta secretion, and up-regulated the expression of IFN-regulated genes in IECs. Those responses were dependent upon activation of the dsRNA binding protein retinoic acid inducible gene I (RIG-I) and the RIG-I interacting protein IFN promoter stimulator-1, but not on dsRNA-activated protein kinase or TLR3, which also were expressed by IECs.
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http://www.ncbi.nlm.nih.gov/pubmed/17911629
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#wasDerivedFrom
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http://purl.org/dc/terms/created
2014-07-03T14:33:12.980+02:00
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