@prefix this: <http://rdf.disgenet.org/nanopublications.trig#NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix sio: <http://semanticscience.org/resource/> .
@prefix ncit: <http://ncicb.nci.nih.gov/xml/owl/EVS/Thesaurus.owl#> .
@prefix lld: <http://linkedlifedata.com/resource/umls/id/> .
@prefix miriam-gene: <http://identifiers.org/ncbigene/> .
@prefix miriam-pubmed: <http://identifiers.org/pubmed/> .
@prefix eco: <http://purl.obolibrary.org/obo/eco.owl#> .
@prefix wi: <http://purl.org/ontology/wi/core#> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix pav: <http://purl.org/pav/2.0/> .
@prefix prv: <http://purl.org/net/provenance/ns#> .
@prefix dcterms: <http://purl.org/dc/terms/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix dgn-np: <http://rdf.disgenet.org/nanopublications.trig#> .
@prefix dgn-gda: <http://rdf.disgenet.org/gene-disease-association.ttl#> .
@prefix dgn-void: <http://rdf.disgenet.org/v2.1.0/void.ttl#> .
dgn-np:NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M130_head {
  this: np:hasAssertion dgn-np:NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M130_assertion ;
    np:hasProvenance dgn-np:NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M130_provenance ;
    np:hasPublicationInfo dgn-np:NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M130_publicationInfo ;
    a np:Nanopublication .
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}
dgn-np:NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M130_assertion {
  miriam-gene:2250 a ncit:C16612 .
  lld:C0001973 a ncit:C7057 .
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    a sio:SIO_001122 .
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dgn-np:NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M130_provenance {
  dgn-np:NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M130_assertion dcterms:description "[These data, coupled with cell and animal model data implicating neuronal signaling in alcohol response, support the conclusion that neuronal signaling is mechanistically involved in alcohols cellular and behavioral effects. Further, these data suggest that genetic variation in these signaling pathways contribute to human variation in alcohol response. Finally, this concordance of the cell, animal, and human findings supports neuronal signaling, particularly glutamate signaling, as a prime target for translational studies to understand and eventually modulate alcohols effects.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
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  dgn-void:source_evidence_literature a eco:ECO_0000212 ;
    rdfs:comment "Gene-disease associations inferred from text-mining the literature."@en ;
    rdfs:label "DisGeNET evidence - LITERATURE"@en .
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dgn-np:NP87697.RAqX297FdJwy4S15L7YFUfrESo3MWo_XUyITWiKnp588M130_publicationInfo {
  this: dcterms:created "2014-10-02T12:32:43+02:00"^^xsd:dateTime ;
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