@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix schem: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix hasPart: . @prefix ProteinComplex: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 hasPart: sub:_2, sub:_3, sub:_4; a ProteinComplex: . sub:_2 geneProductOf: hgnc:12744; a Protein: . sub:_3 geneProductOf: hgnc:7553; a Protein: . sub:_4 geneProductOf: hgnc:6913; a Protein: . sub:_5 occursIn: species:9606; rdf:object sub:_1; rdf:predicate belv:increases; rdf:subject schem:D-Glucose%2C%20anhydrous; a rdf:Statement . sub:assertion rdfs:label "a(SCHEM:\"D-Glucose, anhydrous\") -> complex(p(HGNC:MLXIPL),p(HGNC:MYC),p(HGNC:MAX))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_7; pav:version "20131211" . sub:_6 prov:value "Glucose increased c-Myc abundance and association with its heterodimer partner Max. Manipulations that prevented the formation of a functional c-Myc/Max heterodimer reduced the expression of the L-PK gene. In addition, glucose augmented the binding of ChREBP, c-Myc, and Max to the promoter of the L-PK gene in situ. #[JL] Chrebp is MLXIPL, and is implicated as a glucose-responsive factor. This is basically saying that glucose activates MLXIPL- activation of MYC"; prov:wasQuotedFrom pubmed:17341548 . sub:_7 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:17341548; prov:wasDerivedFrom beldoc:, sub:_6 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:08.897+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }