@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix chebi: . @prefix species: . @prefix occursIn: . @prefix obo: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: obo:CL_0000842, obo:UBERON_0002048, species:9606; rdf:object chebi:15551; rdf:predicate belv:decreases; rdf:subject chebi:18723; a rdf:Statement . sub:assertion rdfs:label "a(CHEBI:nicotine) -| a(SCHEM:Dinoprostone)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "20131211" . sub:_2 prov:value "Nicotine inhibited more efficiently than acetylcholine proinflammatory cytokines, tumor necrosis factor-{alpha} (TNF-{alpha}), interleukin (IL)-1beta, IL-6, and IL-18, but not the anti-inflammatory cytokine IL-10 through a posttranslational mechanism in human peripheral blood mononuclear cells (23, 342).Nicotine also inhibited the production of prostaglandin E2 (PGE2), monocyte inflammatory factor (MIP)-1{alpha} and MIP-1beta in these cells (309, 365). "; prov:wasQuotedFrom pubmed:17615396 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:17615396; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:11.671+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }