@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix chebi: . @prefix RNA: . @prefix mgi: . @prefix geneProductOf: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:96543; a RNA: . sub:_2 occursIn: obo:UBERON_0000947, species:10090; rdf:object sub:_1; rdf:predicate belv:increases; rdf:subject chebi:48432; a rdf:Statement . sub:assertion rdfs:label "a(CHEBI:\"angiotensin II\") -> r(MGI:Il1b)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value "Ang II treatment strikingly induced the expression of proinflammatory cytokines, such as IL-1 and IL-6, as well as intercellular adhesion molecule-1 in the aortas of apoE-deficient mice, whereas p21 deficiency effectively blocked the induction of these molecules by Ang II treatment (Figure 4A; Data Supplement, Figures III and IV)."; prov:wasQuotedFrom pubmed:16908765 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:16908765; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:02.694+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }