@prefix dcterms: . @prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix schem: . @prefix mesh: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 occursIn: obo:CL_0000169, obo:UBERON_0002107, species:9606; rdf:object mesh:D007333; rdf:predicate belv:increases; rdf:subject schem:serum%20insulin; a rdf:Statement . sub:assertion rdfs:label "a(SCHEM:\"serum insulin\") -> bp(MESHPP:\"Insulin Resistance\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_3; pav:version "20131211" . sub:_2 prov:value "Other defects in beta-cell function in type 2 diabetes mellitus include... decreased conversion of proinsulin to insulin hyperinsulinemia inhibits both insulin secretion (39) and action (40), and hyperglycemia can impair both the insulin secretory response to glucose (41) as well as cellular insulin sensitivity (42, 43),"; prov:wasQuotedFrom pubmed:10199747 . sub:_3 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:10199747; prov:wasDerivedFrom beldoc:, sub:_2 . } sub:pubinfo { this: dcterms:created "2014-07-03T14:31:30.059+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }