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a(CHEBI:"carbon monoxide") -| tscript(p(HGNC:HIF1A))
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Approximately 61,000 statements.
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n transient expression studies, both NO and CO inhibited the ability of the hypoxic enhancer we have previously identified to activate gene transcription. Furthermore, electrophoretic mobility shift assays indicated decreased binding of hypoxia-inducible factor 1 (HIF-1) to this enhancer by nuclear proteins isolated from CO-treated cells, although HIF-1 protein levels were unaffected by CO
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Selventa
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2014-07-03T14:34:09.538+02:00
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