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http://rdf.disgenet.org/nanopublications.trig#NP301180.RAnkKnig-A_SdIQjfYhrQ-h5XF79IGNuUhkKYzd0s-HO4
> .
@prefix rdfs: <
http://www.w3.org/2000/01/rdf-schema#
> .
@prefix xsd: <
http://www.w3.org/2001/XMLSchema#
> .
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http://semanticscience.org/resource/
> .
@prefix ncit: <
http://ncicb.nci.nih.gov/xml/owl/EVS/Thesaurus.owl#
> .
@prefix lld: <
http://linkedlifedata.com/resource/umls/id/
> .
@prefix miriam-gene: <
http://identifiers.org/ncbigene/
> .
@prefix miriam-pubmed: <
http://identifiers.org/pubmed/
> .
@prefix eco: <
http://purl.obolibrary.org/obo/eco.owl#
> .
@prefix wi: <
http://purl.org/ontology/wi/core#
> .
@prefix prov: <
http://www.w3.org/ns/prov#
> .
@prefix pav: <
http://purl.org/pav/2.0/
> .
@prefix prv: <
http://purl.org/net/provenance/ns#
> .
@prefix dcterms: <
http://purl.org/dc/terms/
> .
@prefix np: <
http://www.nanopub.org/nschema#
> .
@prefix dgn-np: <
http://rdf.disgenet.org/nanopublications.trig#
> .
@prefix dgn-gda: <
http://rdf.disgenet.org/gene-disease-association.ttl#
> .
@prefix dgn-void: <
http://rdf.disgenet.org/v2.1.0/void.ttl#
> .
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{
this:
np:hasAssertion
dgn-np:NP301180.RAnkKnig-A_SdIQjfYhrQ-h5XF79IGNuUhkKYzd0s-HO4130_assertion
;
np:hasProvenance
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np:hasPublicationInfo
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a
np:Nanopublication
.
dgn-np:NP301180.RAnkKnig-A_SdIQjfYhrQ-h5XF79IGNuUhkKYzd0s-HO4130_assertion
a
np:Assertion
.
dgn-np:NP301180.RAnkKnig-A_SdIQjfYhrQ-h5XF79IGNuUhkKYzd0s-HO4130_provenance
a
np:Provenance
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dgn-np:NP301180.RAnkKnig-A_SdIQjfYhrQ-h5XF79IGNuUhkKYzd0s-HO4130_publicationInfo
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{
miriam-gene:5130
a
ncit:C16612
.
lld:C0205851
a
ncit:C7057
.
dgn-gda:DGN74a8d07cca5f7f0102aad50a19cccfae
sio:SIO_000628
miriam-gene:5130
,
lld:C0205851
;
a
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.
}
dgn-np:NP301180.RAnkKnig-A_SdIQjfYhrQ-h5XF79IGNuUhkKYzd0s-HO4130_provenance
{
dgn-np:NP301180.RAnkKnig-A_SdIQjfYhrQ-h5XF79IGNuUhkKYzd0s-HO4130_assertion
dcterms:description
"[On the basis of the epigenetic phenotypes of CTA genes, we concluded that, first, consistent unmethylated DNA profiles in seminomatous TGCTs imply that methylation may not be the primary control mechanism of programmed gene expression in seminomatous TGCTs, and, second, that nonseminomatous TGCTs might be midway between seminomatous TGCTs and somatic tissues because gene expression in nonseminomatous TGCTs is regulated by methylation in some genes (MAGEA1 and MAGEA3) but not others (SYCP1 and XIST).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
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dgn-void:source_evidence_literature
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sio:SIO_000772
miriam-pubmed:15672408
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prov:wasDerivedFrom
dgn-void:befree-20140225
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prov:wasGeneratedBy
eco:ECO_0000203
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dgn-void:befree-20140225
pav:importedOn
"2014-02-25"^^
xsd:date
.
dgn-void:source_evidence_literature
a
eco:ECO_0000212
;
rdfs:comment
"Gene-disease associations inferred from text-mining the literature."@en ;
rdfs:label
"DisGeNET evidence - LITERATURE"@en .
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dgn-np:NP301180.RAnkKnig-A_SdIQjfYhrQ-h5XF79IGNuUhkKYzd0s-HO4130_publicationInfo
{
this:
dcterms:created
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xsd:dateTime
;
dcterms:rights
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> ;
dcterms:rightsHolder
dgn-void:IBIGroup
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;
prv:usedData
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> , <
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> , <
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pav:createdBy
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