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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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ILK depletion and overexpression respectively decreased and increased the activation of its downstream substrate, PKB/Akt. The pharmacologic inhibition of Akt activity also increased SRF binding to the promoters of smooth muscle specific genes and increased expression of smooth muscle proteins, suggesting that ILK may exert its effects by regulating the activity of Akt.
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