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tscript(complex(SCOMP:"AP-1 Complex")) => r(HGNC:MMP9)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Our results show that, although the deletion construct lacking NF-B (MMP9-608) was less responsive (p 0.05), the construct lacking both NF-B and AP-1 (MMP9-66) had activity close to that of the promoterless pGL3-Basic vector (p 0.001) (Fig. 5C), suggesting that both NF-B and AP-1 play a role in IL-18-mediated MMP9 transcription....These data demonstrate that IL-18 induces MMP9 transcription in SMCs via NF-B and AP-1 transcription factors (Fig. 5).
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Selventa
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2014-07-03T14:32:58.721+02:00
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