@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix go: . @prefix scomp: . @prefix ProteinComplex: . @prefix hasAgent: . @prefix RNA: . @prefix hgnc: . @prefix geneProductOf: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { scomp:AP-1%20Complex a ProteinComplex: . sub:_1 hasAgent: scomp:AP-1%20Complex; a go:0042789 . sub:_2 geneProductOf: hgnc:7176; a RNA: . sub:_3 occursIn: obo:CL_0000192, obo:UBERON_0001621, species:9606; rdf:object sub:_2; rdf:predicate belv:directlyIncreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "tscript(complex(SCOMP:\"AP-1 Complex\")) => r(HGNC:MMP9)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "Our results show that, although the deletion construct lacking NF-B (MMP9-608) was less responsive (p 0.05), the construct lacking both NF-B and AP-1 (MMP9-66) had activity close to that of the promoterless pGL3-Basic vector (p 0.001) (Fig. 5C), suggesting that both NF-B and AP-1 play a role in IL-18-mediated MMP9 transcription....These data demonstrate that IL-18 induces MMP9 transcription in SMCs via NF-B and AP-1 transcription factors (Fig. 5)."; prov:wasQuotedFrom pubmed:16554298 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:16554298; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:32:58.721+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }