@prefix this: <http://rdf.disgenet.org/resource/nanopub/NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix sio: <http://semanticscience.org/resource/> .
@prefix ncit: <http://ncicb.nci.nih.gov/xml/owl/EVS/Thesaurus.owl#> .
@prefix lld: <http://linkedlifedata.com/resource/umls/id/> .
@prefix miriam-gene: <http://identifiers.org/ncbigene/> .
@prefix miriam-pubmed: <http://identifiers.org/pubmed/> .
@prefix eco: <http://purl.obolibrary.org/obo/> .
@prefix wi: <http://purl.org/ontology/wi/core#> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix pav: <http://purl.org/pav/> .
@prefix prv: <http://purl.org/net/provenance/ns#> .
@prefix dcterms: <http://purl.org/dc/terms/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix dgn-np: <http://rdf.disgenet.org/resource/nanopub/> .
@prefix dgn-gda: <http://rdf.disgenet.org/resource/gda/> .
@prefix dgn-void: <http://rdf.disgenet.org/v3.0.0/void/> .
dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_head {
   this: np:hasAssertion dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_assertion ;
    np:hasProvenance dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_provenance ;
    np:hasPublicationInfo dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_publicationInfo ;
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  dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_publicationInfo a np:PublicationInfo .
}
dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_assertion {
   miriam-gene:6093 a ncit:C16612 .
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dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_provenance {
   dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_assertion dcterms:description "[In addition, we have observed the following: (i) HA/CD44-activated Rho kinase (ROK) mediates NHE1 phosphorylation and activity, and (ii) inhibition of ROK or NHE1 activity (by treating cells with a ROK inhibitor, Y27632, or NHE1 blocker, S-(N-ethyl-N-isopropyl) amiloride, respectively) blocks NHE1 phosphorylation/Na(+)-H(+) exchange activity, reduces intracellular acidification, eliminates the acidic environment in the extracellular matrix, and suppresses breast tumor-specific behaviors (e.g.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
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dgn-np:NP674038.RAjH71nbsyLnaua_KsW2sph59hIfYxPsydJnDEPD8OlWo130_publicationInfo {
   this: dcterms:created "2015-08-25T14:44:26+02:00"^^xsd:dateTime ;
    dcterms:rights <http://opendatacommons.org/licenses/odbl/1.0/> ;
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