@prefix dcterms: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix sdis: .
@prefix chebi: .
@prefix species: .
@prefix occursIn: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 occursIn: species:9606;
rdf:object chebi:26523;
rdf:predicate belv:increases;
rdf:subject sdis:peroxisome%20proliferation;
a rdf:Statement .
sub:assertion rdfs:label "path(SDIS:\"peroxisome proliferation\") -> a(CHEBI:\"reactive oxygen species\")" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_3;
pav:version "20131211" .
sub:_2 prov:value "Peroxisomes proliferate in response to external signals triggered by peroxisome-proliferator-activated receptor signalling pathways. Peroxisome-derived oxidative stress as a consequence of this proliferation is increasingly recognized to participate in pathologies ranging from carcinogenesis in rodents to alcoholic and non-alcoholic steatosis hepatitis in humans.";
prov:wasQuotedFrom pubmed:11964148 .
sub:_3 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:11964148;
prov:wasDerivedFrom beldoc:, sub:_2 .
}
sub:pubinfo {
this: dcterms:created "2014-07-03T14:31:48.661+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}