@prefix dcterms: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix chebi: .
@prefix go: .
@prefix atcc: .
@prefix occursIn: .
@prefix species: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 occursIn: atcc:CCL-2.aspx, species:9606;
rdf:object go:0034976;
rdf:predicate belv:increases;
rdf:subject chebi:9516;
a rdf:Statement .
sub:assertion rdfs:label "a(CHEBI:thapsigargin) -> bp(GO:\"response to endoplasmic reticulum stress\")" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_3;
pav:version "1.4" .
sub:_2 prov:value "Lower levels of eIF2alpha phosphorylation in Nck-overexpressing cells were still observed for up to 24 h of treatment with Tn (data not shown) or with the calcium ATPase inhibitor thapsigargin, which also induces ER stress (Tg) (Fig. 1, B and D), demonstrating that Nck overexpression causes a long-lasting inhibition of eIF2alpha Ser51 phosphorylation. ";
prov:wasQuotedFrom pubmed:16835242 .
sub:_3 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:16835242;
prov:wasDerivedFrom beldoc:, sub:_2 .
}
sub:pubinfo {
this: dcterms:created "2014-07-03T14:30:43.936+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}