@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix go: . @prefix RNA: . @prefix mgi: . @prefix geneProductOf: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: mgi:1929938; a RNA: . sub:_2 occursIn: species:10090; rdf:object sub:_1; rdf:predicate belv:increases; rdf:subject go:0034984; a rdf:Statement . sub:assertion rdfs:label "bp(GOBP:\"cellular response to DNA damage stimulus\") -> r(MGI:Perp)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value "# Jubilant: PERP is a direct target of 53, involved in p53-dependent apoptosis. High level PERP expression correlated with the induction of cell death, with 21 percent of cells undergoing apoptosis by 16 hours. Accumulation of high levels of PERP gene expression is dependent on p53, E1A and DNA damage which are all essential for E1A-induced apoptosis in mouse endothelial fibroblasts."; prov:wasQuotedFrom pubmed:10733530 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:10733530; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:31:34.608+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }