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All rights reserved. http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAfcJV7BjAD0MfWxH1pP24ATQquvZ9RDHMCNz9XVMwmGE#_5 http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel http://purl.org/pav/version 20131211 http://www.tkuhn.ch/bel2nanopub/RAfcJV7BjAD0MfWxH1pP24ATQquvZ9RDHMCNz9XVMwmGE#_4 http://www.w3.org/ns/prov#value ecause cross talk between numerous mechanisms of proinflammatory cytokine activation after cigarette smoke exposure likely occurs, we investigated the role of Egr-1 in the absence of CSE. After transfection of Egr-1 (pCMV–Egr-1), secreted IL-1beta was significantly elevated (Figure 7A). 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