dgn-np:NP841307.RAf6gVn2bXQX40eieJZUZIhi79yD1bAuDFUYhQIrHGNCQ130_provenance {
dgn-np:NP841307.RAf6gVn2bXQX40eieJZUZIhi79yD1bAuDFUYhQIrHGNCQ130_assertion dcterms:description "[Based on these findings, we have proposed a hypothesis for the pathogenesis of AIP in the biphasic mechanism of induction and progression. In the early stage, initial response to self-antigens (LF, CA-II, CA-IV, PSTI, or α-fodrin) or molecular mimicry (Helicobacter pylori) is induced by decreased naive regulatory T cells (Tregs), and Th1 cells release proinflammatory cytokines (IFN-γ, IL-1b, IL-2, and TNF-α).]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
wi:evidence dgn-void:source_evidence_literature ;
sio:SIO_000772 miriam-pubmed:20862490 ;
prov:wasDerivedFrom dgn-void:befree-2016 ;
prov:wasGeneratedBy eco:ECO_0000203 .
dgn-void:befree-2016 pav:importedOn "2016-02-19"^^
xsd:date .
dgn-void:source_evidence_literature a eco:ECO_0000212 ;
rdfs:comment "Gene-disease associations inferred from text-mining the literature."@en ;
rdfs:label "DisGeNET evidence - LITERATURE"@en .
}