sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_5 ;
    pav:version "20131211" .
  sub:_4 prov:value "To explore the role of caspase 3 on CSE-induced DNA damage, immunoblotting for caspase 3 and functional evaluation of caspase 3 activity was performed. Under control conditions, BEAS-2B cells produced the 32-kD caspase 3 precursor, and cigarette smoke significantly increased its steady state level in the cell (Figure 6A). In accord with the apoptosis data, cigarette smoke did not convert the precursor form of caspase 3 (32 kD) into the active form (18 kD or smaller fragments), whereas CPT did (Figure 6A)." ;
    prov:wasQuotedFrom pubmed:15845867 .
  sub:_5 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:15845867 ;
    prov:wasDerivedFrom beldoc: , sub:_4 .
}