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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.nanopub.org/nschema#Nanopublication
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http://www.tkuhn.ch/bel2nanopub/RAdoCk9YXT9wOf2xwzS8YduUTZAoAO8gNwjOljD9iws9Y#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.informatics.jax.org/marker/MGI:96545
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://purl.obolibrary.org/obo/BFO_0000066
http://disease-ontology.org/term/DOID:1936
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
http://purl.bioontology.org/ontology/MSH/D050197
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http://www.w3.org/2000/01/rdf-schema#label
p(MGI:Il1r1) -> path(MESHD:Atherosclerosis)
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
http://purl.org/dc/elements/1.1/description
Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
http://purl.org/dc/elements/1.1/title
BEL Framework Large Corpus Document
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http://www.w3.org/ns/prov#value
animal models, with reduced atherosclerosis observed in IL-1b [41] and IL-1 receptor-deficient mice [11], while IL-1ra-deficient mice exhibit increased neointima formation [42]. IL-1Ra administration to Apo E-deficient mice or porcine arterial injury models reduced neointima formation significantly [11,43] as well as reducing vascular remodelling following acute myocardial infarction in the rat [44].
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
http://www.ncbi.nlm.nih.gov/pubmed/19309349
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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2014-07-03T14:33:33.578+02:00
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http://orcid.org/0000-0002-1267-0234