@prefix this: . @prefix rdfs: . @prefix xsd: . @prefix sio: . @prefix ncit: . @prefix lld: . @prefix miriam-gene: . @prefix miriam-pubmed: . @prefix eco: . @prefix wi: . @prefix prov: . @prefix pav: . @prefix prv: . @prefix dcterms: . @prefix np: . @prefix dgn-np: . @prefix dgn-gda: . @prefix dgn-void: . dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_head { this: np:hasAssertion dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_assertion; np:hasProvenance dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_provenance; np:hasPublicationInfo dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_publicationInfo; a np:Nanopublication . dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_assertion a np:Assertion . dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_provenance a np:Provenance . dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_publicationInfo a np:PublicationInfo . } dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_assertion { miriam-gene:9636 a ncit:C16612 . lld:C0000768 a ncit:C7057 . dgn-gda:DGN110dba6af6f89aad7571cf2ffd02b1fa sio:SIO_000628 miriam-gene:9636, lld:C0000768; a sio:SIO_001121 . } dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_provenance { dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_assertion dcterms:description "[The genes functions were related to a diverse cellular process including: (1) most of these genes were associated with CD4+ T cells functions, particularly related to cellular developments; (2) Ras pathway genes as RANBP10, GMIP, RASGRP2 and ARL5 might be responsible for the abnormal development of CD4+ T cells of SLE; (3) HIG2, TCF7, KHSRP, WWP1, SMAD3, TLK2, AES, CCNI and PIM2 belong to Wnt/beta-catenin way, they could play roles in modulating proliferation and differentiation of T lymphocytes; (4) uncertain viral infections may initiate autoimmunity because high levels expression genes were detected in T4-1s such as TRIM22, IER2, ABCE1, DUT, G1P2, G1P3, HNRPUL1, EVER2, IFNAR1, TNFSF14, TMP21 and PVRL2; and (5) apoptosis relating genes as EIF3S8, SH3BGRL3, GPX4, TOSO, PFDN5, BIN1, XIAPAF1, TEGT and CUGBP2 may contribute to over uploading of selfantigens in SLE cells.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en; wi:evidence dgn-void:source_evidence_literature; sio:SIO_000772 miriam-pubmed:16143398; prov:wasDerivedFrom dgn-void:befree-2016; prov:wasGeneratedBy eco:ECO_0000203 . dgn-void:befree-2016 pav:importedOn "2016-02-19"^^xsd:date . dgn-void:source_evidence_literature a eco:ECO_0000212; rdfs:comment "Gene-disease associations inferred from text-mining the literature."@en; rdfs:label "DisGeNET evidence - LITERATURE"@en . } dgn-np:NP512228.RAclx1WBrKuqxCH-JhQ39SUxdWCk7bSceSIll1LZ6FhAU130_publicationInfo { this: dcterms:created "2016-05-13T12:45:37+02:00"^^xsd:dateTime; dcterms:rights ; dcterms:rightsHolder dgn-void:IBIGroup; dcterms:subject sio:SIO_000983; prv:usedData dgn-void:disgenetv3.0rdf; pav:authoredBy , , , , ; pav:createdBy ; pav:version "v4.0.0.0" . dgn-void:disgenetv3.0rdf pav:version "v4.0.0" . }