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path(MESHD:Hyperoxia) -| sec(p(MGI:Vegfa))
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Approximately 61,000 statements.
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BEL Framework Large Corpus Document
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Successful treatment of established human tumors, however, might require not only prevention of further angiogenesis but also destruction of already existing tumor blood vessels to reduce the already existing tumor mass. In line with this, VEGF has been shown to not only act as an angiogenic factor but also as a survival factor for endothelial cells and newly formed blood vessels. In the developing retina, hyperoxia-induced vessel regression has been correlated with inhibition of glial VEGF release (5).
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Selventa
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2014-07-03T14:32:12.924+02:00
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