@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix species: . @prefix occursIn: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:11935; a Protein: . sub:_2 geneProductOf: hgnc:5438; a Protein: . sub:_3 occursIn: species:9606; rdf:object sub:_2; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:CD40LG) -> p(HGNC:IFNG)" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "Treatment of immature monocyte-derived dendritic cells (iMDDCs) with a combination of all three TNF family molecules did not always enhance CTL responses (activity was measured by checking the increase in intracellular IFN gamma production) above those with CD40LT treatment alone. Thus, CD40LT, TNF-alpha, or RANKLT conditioning of iMDDCs could expand HIV-1-specific memory CTL with the greatest expansions observed with CD40LT."; prov:wasQuotedFrom pubmed:12574344 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12574344; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:31:58.754+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }