@prefix this: <http://rdf.disgenet.org/nanopublications.trig#NP185253.RAcNGgWYiUQMIjHzmpjzypgx9EFvi5TZnv-TFwQlMbilo> .
@prefix rdfs: <http://www.w3.org/2000/01/rdf-schema#> .
@prefix xsd: <http://www.w3.org/2001/XMLSchema#> .
@prefix sio: <http://semanticscience.org/resource/> .
@prefix ncit: <http://ncicb.nci.nih.gov/xml/owl/EVS/Thesaurus.owl#> .
@prefix lld: <http://linkedlifedata.com/resource/umls/id/> .
@prefix miriam-gene: <http://identifiers.org/ncbigene/> .
@prefix miriam-pubmed: <http://identifiers.org/pubmed/> .
@prefix eco: <http://purl.obolibrary.org/obo/eco.owl#> .
@prefix wi: <http://purl.org/ontology/wi/core#> .
@prefix prov: <http://www.w3.org/ns/prov#> .
@prefix pav: <http://purl.org/pav/2.0/> .
@prefix prv: <http://purl.org/net/provenance/ns#> .
@prefix dcterms: <http://purl.org/dc/terms/> .
@prefix np: <http://www.nanopub.org/nschema#> .
@prefix dgn-np: <http://rdf.disgenet.org/nanopublications.trig#> .
@prefix dgn-gda: <http://rdf.disgenet.org/gene-disease-association.ttl#> .
@prefix dgn-void: <http://rdf.disgenet.org/v2.1.0/void.ttl#> .
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  this: np:hasAssertion dgn-np:NP185253.RAcNGgWYiUQMIjHzmpjzypgx9EFvi5TZnv-TFwQlMbilo130_assertion ;
    np:hasProvenance dgn-np:NP185253.RAcNGgWYiUQMIjHzmpjzypgx9EFvi5TZnv-TFwQlMbilo130_provenance ;
    np:hasPublicationInfo dgn-np:NP185253.RAcNGgWYiUQMIjHzmpjzypgx9EFvi5TZnv-TFwQlMbilo130_publicationInfo ;
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dgn-np:NP185253.RAcNGgWYiUQMIjHzmpjzypgx9EFvi5TZnv-TFwQlMbilo130_assertion {
  miriam-gene:472 a ncit:C16612 .
  lld:C1704437 a ncit:C7057 .
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dgn-np:NP185253.RAcNGgWYiUQMIjHzmpjzypgx9EFvi5TZnv-TFwQlMbilo130_provenance {
  dgn-np:NP185253.RAcNGgWYiUQMIjHzmpjzypgx9EFvi5TZnv-TFwQlMbilo130_assertion dcterms:description "[These results (i) confirm findings by others suggesting assignment of the ATD and ATC genes to chromosome 11, (ii) demonstrate that several genes can modify the cellular radiation response when they are taken out of their normal genomic context and/or control, and (iii) indicate that the RDS phenotype and the enhanced cell killing in A-T are independent pleiotropic features resulting from the primary mutations in A-T. Also, our findings underscore that, in establishing cDNAs as candidate genes for A-T, microcell-mediated chromosome transfer studies are needed to exclude nonspecific correcting effects of these candidate cDNA genes.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ;
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dgn-np:NP185253.RAcNGgWYiUQMIjHzmpjzypgx9EFvi5TZnv-TFwQlMbilo130_publicationInfo {
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