sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_8 ;
    pav:version "20131211" .
  sub:_7 prov:value "CAPRI-knockout mouse. Cells from this animal have the predicted hyperactivation of Ras signalling, which is consistent with the loss of an important Ras-GAP. Interestingly, Zhang et al. [40] also noted a decrease in the activation of the small GTPases Rac and Cdc42. This effect appeared not to arise through the hyperactivation of Ras, nor was it a consequence of CAPRI functioning as a Cdc42/Rac GEF, rather the authors show that CAPRI, via its Ras-GAP domain, can bind Rac and Cdc42 directly and act as a scaffold for presenting these GTPases to an unidentified GEF. " ;
    prov:wasQuotedFrom pubmed:17052212 .
  sub:_8 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:17052212 ;
    prov:wasDerivedFrom beldoc: , sub:_7 .
}