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http://www.nanopub.org/nschema#hasPublicationInfo
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAa467ZxbHleKoMNgHQ_j9ViAzhA8zHo6oHfBqPGsH5pA#_1
http://purl.obolibrary.org/obo/RO_0002204
http://rgd.mcw.edu/rgdweb/report/gene/main.html?id=621320
http://www.tkuhn.ch/bel2nanopub/RAa467ZxbHleKoMNgHQ_j9ViAzhA8zHo6oHfBqPGsH5pA#_1
http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_33697
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
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a(CHEBI:budesonide) -| (a(CHEBI:lipopolysaccharide) -> r(RGD:Mmp9))
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http://www.w3.org/2000/01/rdf-schema#label
a(CHEBI:lipopolysaccharide) -> r(RGD:Mmp9)
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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Exposure to aerosolized LPS resulted in a significant increase in gene expression of a range of pro-inflammatory mediators in the lung tissue (Table 1). Treatment with resveratrol did not impact on any of the LPS-induced increases in gene expression measured. The positive control, budesonide, however caused dose-related inhibition of all of the pro-inflammatory genes measured (Table 1).
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Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://purl.org/dc/terms/created
2014-07-03T14:32:40.582+02:00
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