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http://www.tkuhn.ch/bel2nanopub/RA_Ys8D0WgsxTJOkGtxhgbe5s6FnkONoRWME4RhHir6OY#_1
http://semanticscience.org/resource/SIO_000139
http://www.tkuhn.ch/bel2nanopub/RA_Ys8D0WgsxTJOkGtxhgbe5s6FnkONoRWME4RhHir6OY#_2
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a(SCHEM:"serum glucose") -| tscript(p(HGNC:XBP1))
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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cis-regulatory element located at nt +1614-1621 (5'-TGACGCAA-3') is responsible for activation of the C/EBPbeta gene. Electrophoresis mobility shift analysis revealed that proteins are bound to this element and that the amount of binding is increased following glucose deprivation. This element is homologous to a previously reported mammalian unfolded protein response element that binds XBP-1. Consistent with those data, overexpression of XBP-1 caused an increase in transcription that was mediated by the C/EBPbeta mammalian unfolded protein response element.
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Selventa
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2014-07-03T14:30:22.754+02:00
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