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kin(p(SFAM:"MAPK p38 Family")) -> tscript(p(MGI:Atf2))
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Approximately 61,000 statements.
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However, TNF treatment did result in activation of the transcription factor, ATF-2, a primary downstream target of p38 MAP kinase. Use of the p38 MAP kinase inhibitors SB202190 and SB203580 did not interfere with the ability of TNF to activate ATF-2, suggesting that either the gamma isoform of p38 MAP kinase or a p38-independent pathway was utilized by TNF to increase the phosphorylated fraction of ATF-2.
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