. . . . . . . . . . . . . "a(CHEBI:lipopolysaccharide) -> r(MGI:Saa1)" . "Approximately 61,000 statements." . "Copyright (c) 2011-2012, Selventa. All rights reserved." . "BEL Framework Large Corpus Document" . . "20131211" . "Meng et al. [23] conducted a study to determine whether co-exposure to smoke and LPS enhanced inflammatory response and other pathological effects related to COPD development in mice (Table 3). The most up-regulated genes in the three groups were Mmp12 (matrix metalloproteinase 12, 18.2-fold) in the smoke group, serum amyloid A1 in the LPS (242.4-fold), and LPS–smoke group (167.7- fold). Marco (macrophage receptor with collagenous structure) was also up-regulated by LPS (14.8-fold) and LPS–smoke (25.5-fold) groups. Noxo1 (NADPH oxidase organizer 1) was induced by all three treatment groups." . . "Selventa" . . . . "2014-07-03T14:33:12.548+02:00"^^ . . .