sub:provenance {
  beldoc: dce:description "Approximately 61,000 statements." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
    dce:title "BEL Framework Large Corpus Document" ;
    pav:authoredBy sub:_4 ;
    pav:version "20131211" .
  sub:_3 prov:value "Meng et al. [23] conducted a study to determine whether co-exposure to smoke and LPS enhanced inflammatory response and other pathological effects related to COPD development in mice (Table 3). The most up-regulated genes in the three groups were Mmp12 (matrix metalloproteinase 12, 18.2-fold) in the smoke group, serum amyloid A1 in the LPS (242.4-fold), and LPS–smoke group (167.7- fold). Marco (macrophage receptor with collagenous structure) was also up-regulated by LPS (14.8-fold) and LPS–smoke (25.5-fold) groups. Noxo1 (NADPH oxidase organizer 1) was induced by all three treatment groups." ;
    prov:wasQuotedFrom pubmed:17888717 .
  sub:_4 rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:17888717 ;
    prov:wasDerivedFrom beldoc: , sub:_3 .
}