. . . . . . . "[Altogether, our data reveal a previously unrecognized role of GPR84 in maintaining fully developed AML by sustaining aberrant ?-catenin signaling in LSCs, and suggest that targeting the oncogenic GPR84/?-catenin signaling axis may represent a novel therapeutic strategy for AML.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en . . . . . "2017-02-19"^^ . . "Gene-disease associations inferred from text-mining the literature."@en . "DisGeNET evidence - LITERATURE"@en . "2017-10-17T13:18:34+02:00"^^ . . . . . . . . . . . "v5.0.0.0" . "v5.0.0" .