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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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1.4
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(Jubilant) Pretreatment with C3 transferase exoenzyme, an inhibitor of Rho small GTP binding proteins (Rho A, B and C) inhibited the bradykinin induced NFkB activity. Treatment with calpain I or PDTC completely inhibited the DNA binding activity of NFkB. Transfection of cell with dominant negative RhoA-T19N inhibited the bradykinin induced NFkB activity. A dominant-negative form of RhoA (T19N) inhibited BK-stimulated reporter gene expression in a dose-dependent and kappaB-dependent manner.
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Selventa
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2014-07-03T14:30:59.496+02:00
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