sub:provenance {
beldoc: dce:description "Approximately 61,000 statements." ;
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved." ;
dce:title "BEL Framework Large Corpus Document" ;
pav:authoredBy sub:_4 ;
pav:version "20131211" .
sub:_3 prov:value "MuRF1 -/- and MAFbx -/- mice appear phenotypically normal. However, under atrophy conditions, significantly less muscle mass is lost in either MuRF1 -/- or MAFbx -/- animals in comparison with control littermates [11]. Myostatin However, when muscle obtained from myostatin -/- animals was analyzed, it was shown to be larger as a result of an increase in the number of muscle fibers, and not as a result of hypertrophy. Therefore, it was thought that myostatin acts in a way that is distinct from the activation of atrophy or the inhibition of hypertrophy; that it simply blocks the proliferation of muscle precursors, thus decreasing muscle mass." ;
prov:wasQuotedFrom pubmed:12928036 .
sub:_4 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:12928036 ;
prov:wasDerivedFrom beldoc: ,
sub:_3 .
}