@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix go: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: hgnc:11892; a Protein: . sub:_2 occursIn: obo:CL_0002618, species:9606; rdf:object go:0055133; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(HGNC:TNF) -| bp(GOBP:\"DNA replication\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_4; pav:version "20131211" . sub:_3 prov:value "Tumour necrosis factor-a (TNF-a), which was used to induce cell arrest in the G1 phase of HUVEC (positive control) [20], decreased the incorporation of [3H]thymidine by 60% and maintained 90% of cells in the G1 phase in comparison with control cells (65%) (Table 1), whereas DHEA decreased the incorporation of [3H]thymidine by 64% and induced arrest in the G1 phase of the cell cycle in 88% of the cells, similarly to TNF-a-stimulated cells."; prov:wasQuotedFrom pubmed:15752352 . sub:_4 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:15752352; prov:wasDerivedFrom beldoc:, sub:_3 . } sub:pubinfo { this: dct:created "2014-07-03T14:32:41.254+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }