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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
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http://www.tkuhn.ch/bel2nanopub/RAXmgHhD646Y2MvncNjXiwmCoOnw6A_3hn9IDVRNTLpW4#_1
http://purl.obolibrary.org/obo/RO_0002204
http://www.genenames.org/cgi-bin/gene_symbol_report?hgnc_id=24944
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http://www.w3.org/1999/02/22-rdf-syntax-ns#type
http://www.ebi.ac.uk/chebi/searchId.do?chebiId=CHEBI_36080
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http://purl.obolibrary.org/obo/BFO_0000066
http://purl.obolibrary.org/obo/UBERON_0002367
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http://purl.obolibrary.org/obo/BFO_0000066
http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?id=9606
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http://www.w3.org/1999/02/22-rdf-syntax-ns#object
http://purl.bioontology.org/ontology/MSH/D017209
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http://www.w3.org/2000/01/rdf-schema#label
p(HGNC:DDIT4) -| bp(MESHPP:Apoptosis)
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http://resource.belframework.org/belframework/20131211/knowledge/large_corpus.bel
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Approximately 61,000 statements.
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Copyright (c) 2011-2012, Selventa. All rights reserved.
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BEL Framework Large Corpus Document
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http://www.w3.org/ns/prov#value
Reduced REDD1 levels can sensitize cells towards apoptosis, whereas elevated levels of REDD1 induced by hypoxia or overexpression desensitize cells to apoptotic stimuli. Taken together our data designate REDD1 as a novel target for therapeutic intervention in prostate cancer.
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http://www.w3.org/ns/prov#wasQuotedFrom
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http://www.w3.org/2000/01/rdf-schema#label
Selventa
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http://www.w3.org/ns/prov#hadPrimarySource
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http://www.w3.org/ns/prov#wasDerivedFrom
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http://www.tkuhn.ch/bel2nanopub/RAXmgHhD646Y2MvncNjXiwmCoOnw6A_3hn9IDVRNTLpW4#pubinfo
http://www.tkuhn.ch/bel2nanopub/RAXmgHhD646Y2MvncNjXiwmCoOnw6A_3hn9IDVRNTLpW4
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2014-07-03T14:32:36.469+02:00
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http://orcid.org/0000-0001-6818-334X
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http://orcid.org/0000-0002-1267-0234