. . . . . . . . . . . . . . . . . . . "kin(p(MGI:Akt2)) -> tport(p(MGI:Slc2a4))" . "Approximately 61,000 statements." . "Copyright (c) 2011-2012, Selventa. All rights reserved." . "BEL Framework Large Corpus Document" . . "20131211" . "Furthermore, skeletal myotube hypertrophy induced by IG1 could be inhibited by a pharmacological inhibitor of PI3K . Therefore, PI3K activity is necessary for IGF1 to induce hypertrophy, and its activation is sufficient to induce hypertrophy. For example, knockout mice that are Akt2 are normal in size but have a diabetic phenotype because of insulin resistance . The PI3K Akt2 pathway has been linked to glucose transport via the Glut4 transporter" . . "Selventa" . . . . "2014-07-03T14:32:07.434+02:00"^^ . . .