@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix go: . @prefix Protein: . @prefix hgnc: . @prefix geneProductOf: . @prefix hasAgent: . @prefix chebi: . @prefix obo: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 hasAgent: sub:_2; a go:0003824 . sub:_2 geneProductOf: hgnc:11180; a Protein: . sub:_3 occursIn: obo:CL_0000115, obo:UBERON_0004535, species:9606; rdf:object chebi:26523; rdf:predicate belv:decreases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "cat(p(HGNC:SOD2)) -| a(CHEBI:\"reactive oxygen species\")" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_5; pav:version "20131211" . sub:_4 prov:value "Incubation of endothelial cells with high concentrations of FFAs, similar to those found in insulin-resistant subjects, has been shown to increase O2 . production several fold.153. The increase in ROS production was inhibited by overexpression of either UCP-1 or SOD2, which indicates that mitochondrial ETC is the source of FFA-induced ROS production."; prov:wasQuotedFrom pubmed:17332437 . sub:_5 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:17332437; prov:wasDerivedFrom beldoc:, sub:_4 . } sub:pubinfo { this: dct:created "2014-07-03T14:33:08.853+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }